INT157905

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Context Info
Confidence 0.31
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.91
Pain Relevance 0.25

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (TRPV2) plasma membrane (TRPV2) intracellular (TRPV2)
cytoplasm (TRPV2)
Anatomy Link Frequency
neuronal 1
TRPV2 (Homo sapiens)
Pain Link Frequency Relevance Heat
TRP channel 4 92.48 High High
Pain 1 88.40 High High
Disease Link Frequency Relevance Heat
Myocardial Infarction 1 99.52 Very High Very High Very High
Coronary Heart Disease 2 99.16 Very High Very High Very High
Frailty 1 97.52 Very High Very High Very High
Disease 2 93.84 High High
Heart Disease 3 91.16 High High
Pain 1 88.40 High High
Death 1 82.84 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Evidence suggests that (a) upregulation of TRPC channels is involved in the development of cardiac hypertrophy and heart failure; (b) TRPC1, TRPC6, and TRPV2 play a role in the pathogenesis of cardiomyopathy associated with muscular dystrophy; (c) TRPC6 or TRPM4 is involved in the delayed after-depolarization; (d) TRPP2 is involved in the normal development of the interventricular and interatrial septa; and (e) neuronal TRPV1 acts as a detector of pain-producing stimuli.
TRPV2 Binding (play) of in neuronal associated with pain, frailty, coronary heart disease and myocardial infarction
1) Confidence 0.31 Published 2009 Journal Circ. J. Section Abstract Doc Link 19202304 Disease Relevance 0.91 Pain Relevance 0.25

General Comments

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