INT15870

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Context Info
Confidence 0.25
First Reported 1991
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 6
Total Number 6
Disease Relevance 3.22
Pain Relevance 0.85

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
myocardium 1
liver 1
margin 1
spikes 1
neurons 1
PSS (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 4 96.80 Very High Very High Very High
Potency 2 91.00 High High
Pyramidal cell 2 86.96 High High
anesthesia 3 86.52 High High
halothane 1 86.08 High High
Sicca syndrome 2 81.48 Quite High
rheumatoid arthritis 6 77.48 Quite High
Hippocampus 4 75.00 Quite High
ischemia 1 75.00 Quite High
corticosteroid 2 61.12 Quite High
Disease Link Frequency Relevance Heat
Salivary Gland Disease 1 100.00 Very High Very High Very High
Cryoglobulinemia 146 99.16 Very High Very High Very High
Hepatitis C Virus Infection 84 98.16 Very High Very High Very High
Sunburn 2 98.12 Very High Very High Very High
Infection 25 98.00 Very High Very High Very High
Hypersensitivity Vasculitis 7 97.16 Very High Very High Very High
Syndrome 45 92.52 High High
Lymphatic System Cancer 17 91.76 High High
Purpura 13 88.56 High High
Arthralgia 7 88.20 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Targeting PSs specifically could not only avoid such side-effects, it could greatly improve PDT's therapeutic margin.
Regulation (Targeting) of PSs in margin
1) Confidence 0.25 Published 2008 Journal Sci Prog Section Abstract Doc Link 18853576 Disease Relevance 0.26 Pain Relevance 0.12
Results indicate that a significant change in PSS (from 7.46% +/- 2.14%, mean +/- SEM to 17.74% +/- 3.31%; P less than 0.00001) was associated with a reduction in coronary blood flow from 81.48 +/- 8.85 to 56.94 +/- 7.12 mL/min/100 g tissue (P less than 0.0012), but lactate extraction across the myocardium did not change (10.54% +/- 3.20% to 12.17% +/- 2.43%).
Regulation (change) of PSS in myocardium
2) Confidence 0.23 Published 1991 Journal J. Cardiothorac. Vasc. Anesth. Section Abstract Doc Link 1768816 Disease Relevance 0.07 Pain Relevance 0.17
The results imply that the effects of sevoflurane on PSs are greater in DG than in CA1 neurons, that sevoflurane enhances the PPF in both CA1 and DG, and that the actions of sevoflurane are not similar to those of other volatile or intravenous anesthetics previously reported in hippocampal preparations.
Regulation (effects) of PSs in neurons
3) Confidence 0.23 Published 1997 Journal Anesth. Analg. Section Abstract Doc Link 9249125 Disease Relevance 0 Pain Relevance 0.17
We studied the effects of sevoflurane on population spikes (PSs) in two synaptic pathways in rat hippocampal slices.
Regulation (effects) of PSs in spikes
4) Confidence 0.23 Published 1997 Journal Anesth. Analg. Section Abstract Doc Link 9249125 Disease Relevance 0 Pain Relevance 0.08
This has implications for the future engineering of scFv carriers for PDT, in order to control the number and function of the PSs that can be coupled.
Regulation (control) of PSs
5) Confidence 0.21 Published 2008 Journal Sci Prog Section Abstract Doc Link 18853576 Disease Relevance 0.20 Pain Relevance 0.11
However, a careful patient clinical assessment is usually sufficient for a correct diagnosis in the large majority of cases by considering some important findings: histopathological alterations of salivary glands and specific autoantibody pattern (anti-RoSSA/LaSSB) of pSS are rarely found in MC patients; conversely, HCV infection, cutaneous leukocytoclastic vasculitis, and visceral organ involvement (renal, liver) are seldom recorded in primary Sjögren's syndrome (Fig. 7).
Regulation (alterations) of pSS in liver associated with cryoglobulinemia, syndrome, salivary gland disease, hypersensitivity vasculitis, hepatitis c virus infection and infection
6) Confidence 0.05 Published 2008 Journal Orphanet J Rare Dis Section Body Doc Link PMC2569912 Disease Relevance 2.69 Pain Relevance 0.20

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