INT159665

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.25
First Reported 2008
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 3
Disease Relevance 0.27
Pain Relevance 0.34

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Camk1g) plasma membrane (Camk1g) cytoplasm (Camk1g)
Camk1g (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Hippocampus 3 97.64 Very High Very High Very High
antidepressant 4 75.00 Quite High
fluoxetine 1 74.04 Quite High
Potency 2 64.04 Quite High
hyperexcitability 8 9.72 Low Low
Neurotransmitter 12 5.00 Very Low Very Low Very Low
bDMF 10 5.00 Very Low Very Low Very Low
Dopamine 8 5.00 Very Low Very Low Very Low
monoamine 6 5.00 Very Low Very Low Very Low
Serotonin 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Urological Neuroanatomy 17 85.92 High High
Toxicity 2 75.24 Quite High
Drug Induced Neurotoxicity 6 58.88 Quite High
Osteogenic Sarcomas 4 17.36 Low Low
Poisoning 8 5.00 Very Low Very Low Very Low
Neuroblastoma 4 5.00 Very Low Very Low Very Low
Repression 4 5.00 Very Low Very Low Very Low
Shock 4 5.00 Very Low Very Low Very Low
Hypoxia 2 5.00 Very Low Very Low Very Low
Decapitation 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Dose-dependent changes in the transcription of several genes (Camk1g, Ddc, Gpd3, c-fos and Egr1) were discovered and successfully confirmed.
Regulation (changes) of Camk1g
1) Confidence 0.25 Published 2008 Journal BMC Genomics Section Body Doc Link PMC2626604 Disease Relevance 0.19 Pain Relevance 0.03
(Camk1g) and dopa decarboxylase (Ddc) were commonly affected by both compounds indicating that for these genes there was no differences in the changes in expression elicited by equipotent doses of either pyrethroid.
Regulation (affected) of Camk1g
2) Confidence 0.25 Published 2008 Journal BMC Genomics Section Body Doc Link PMC2626604 Disease Relevance 0 Pain Relevance 0
The results also showed that, contrary to the activatory role of MAP-ERKs and CaM kinase IV, nuclear alphaCaM kinase II was inactivated in parallel with activation of CREB.
Regulation (role) of CaM kinase IV
3) Confidence 0.07 Published 2009 Journal Int. J. Neuropsychopharmacol. Section Abstract Doc Link 19400982 Disease Relevance 0.09 Pain Relevance 0.31

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox