INT160225

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Context Info
Confidence 0.28
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 2.18
Pain Relevance 0.25

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Acot1, Ache) nuclear envelope (Ache) mitochondrion (Acot1)
Golgi apparatus (Ache) lipid metabolic process (Acot1) cytosol (Acot1)
Anatomy Link Frequency
neurons 1
Acot1 (Rattus norvegicus)
Ache (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Hippocampus 2 82.08 Quite High
Neurotransmitter 2 61.36 Quite High
headache 2 57.80 Quite High
Disease Link Frequency Relevance Heat
Hypoxia 18 99.76 Very High Very High Very High
Cognitive Disorder 8 75.36 Quite High
Vomiting 2 58.24 Quite High
Headache 2 57.80 Quite High
Dizziness 2 56.56 Quite High
Sleep Disorders 2 56.12 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The AChEIs were able to improve the cholinergic activity by restoring the level of ACh by blocking the AChE activity.
AChEIs Negative_regulation (blocking) of AChE
1) Confidence 0.28 Published 2009 Journal Behav. Brain Res. Section Abstract Doc Link 19446892 Disease Relevance 1.18 Pain Relevance 0.13
In addition, the AChEIs also prevented neurodegeneration by reducing the AChE level in cortical and hippocampal neurons.
AChEIs Negative_regulation (reducing) of AChE in neurons
2) Confidence 0.24 Published 2009 Journal Behav. Brain Res. Section Abstract Doc Link 19446892 Disease Relevance 1.00 Pain Relevance 0.12

General Comments

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