INT160761

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Context Info
Confidence 0.01
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.07
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

intracellular (Cma1) cytoplasm (ENG) peptidase activity (Cma1)
extracellular space (ENG) extracellular region (Cma1) cell motility (ENG)
Anatomy Link Frequency
brain 2
Cma1 (Rattus norvegicus)
ENG (Homo sapiens)
Pain Link Frequency Relevance Heat
Opioid 1 50.00 Quite Low
Endogenous opioid 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 1 65.20 Quite High
Anxiety Disorder 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
CONCLUSION: CMA is effective in increasing ALLO and beta-END in selective brain areas showing a specific pattern of interaction with brain function, different compared to progesterone or to other synthetic progestins.
CMA Regulation (effective) of Positive_regulation (increasing) of beta-END in brain
1) Confidence 0.01 Published 2009 Journal Contraception Section Body Doc Link 19501216 Disease Relevance 0.07 Pain Relevance 0

General Comments

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