INT161005

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Context Info
Confidence 0.15
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.52
Pain Relevance 0.37

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cytoplasm (Gfap, St13) unfolded protein binding (St13) protein folding (St13)
cytosol (St13) structural molecule activity (Gfap) extracellular matrix organization (Gfap)
Anatomy Link Frequency
caudate-putamen 1
Gfap (Mus musculus)
St13 (Mus musculus)
Pain Link Frequency Relevance Heat
Dopamine 5 98.96 Very High Very High Very High
Enkephalin 4 98.78 Very High Very High Very High
Substantia nigra 1 96.46 Very High Very High Very High
Disease Link Frequency Relevance Heat
Loss Of Sense Of Smell 1 93.04 High High
Parkinson's Disease 2 83.24 Quite High
Disease 6 83.00 Quite High
Gliosis 1 41.04 Quite Low
Pathologic Processes 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We evaluated behavioral deficits (olfactory and motor impairment), neurodegeneration (loss of tyrosine hydroxylase (TH)-positive cells in the substantia nigra pars compacta, SNc), biochemical markers of functional impairment in the caudate-putamen (CPu) (striatal enkephalin mRNA, DA and DOPAC levels), and glial reactivity (CD11b and GFAP immunoreactivity in the SNc and CPu) at progressive time-points (after 1, 3, 7, and 10 administrations of MPTPp).
GFAP Binding (immunoreactivity) of SNc in caudate-putamen associated with dopamine, substantia nigra and enkephalin
1) Confidence 0.15 Published 2009 Journal Neurotox Res Section Abstract Doc Link 19526289 Disease Relevance 0.52 Pain Relevance 0.37

General Comments

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