INT161305

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Context Info
Confidence 0.25
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.25
Pain Relevance 0.83

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (NFKB1) mitochondrion (NFKB1) cell-cell signaling (IL2)
cytoplasm (NFKB1) cytosol (NFKB1) extracellular space (IL2)
Anatomy Link Frequency
T lymphocytes 1
NFKB1 (Homo sapiens)
IL2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Endogenous opioid 1 96.88 Very High Very High Very High
Morphine 3 96.36 Very High Very High Very High
Opioid 6 93.12 High High
Pain 1 76.92 Quite High
mu opioid receptor 1 42.00 Quite Low
Disease Link Frequency Relevance Heat
Acquired Immune Deficiency Syndrome Or Hiv Infection 1 86.20 High High
Cancer 1 85.88 High High
Pain 1 76.92 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We demonstrated that morphine and the endogenous opioid beta-endorphin inhibited the transcription of IL-2 in activated human T lymphocytes as well as the activation of the transcription factors AP-1, NFAT, and NF-kappaB, which transactivate IL-2.
NF-kappaB Positive_regulation (transactivate) of IL-2 in T lymphocytes associated with endogenous opioid and morphine
1) Confidence 0.25 Published 2009 Journal J. Immunol. Section Abstract Doc Link 19561113 Disease Relevance 0.25 Pain Relevance 0.83

General Comments

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