INT161670

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Context Info
Confidence 0.14
First Reported 2009
Last Reported 2009
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.73
Pain Relevance 1.41

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Mapk1) mitochondrion (Mapk1) protein complex (Mapk1)
cytoplasm (Mapk1) cytosol (Mapk1) signal transduction (Mapk1)
Anatomy Link Frequency
spinal cord 2
H2-M3 (Mus musculus)
Mapk1 (Mus musculus)
Pain Link Frequency Relevance Heat
allodynia 8 100.00 Very High Very High Very High
cytokine 1 97.40 Very High Very High Very High
Spinal cord 3 95.52 Very High Very High Very High
Glutamate receptor 1 79.00 Quite High
Pain management 1 78.00 Quite High
Morphine 6 75.20 Quite High
intrathecal 1 75.00 Quite High
primary afferent fibers 1 73.32 Quite High
Dynorphin 1 72.08 Quite High
Glutamate 1 71.48 Quite High
Disease Link Frequency Relevance Heat
Neuropathic Pain 8 100.00 Very High Very High Very High
Nociception 1 68.88 Quite High
Myoclonus 1 50.96 Quite High
Hyperalgesia 1 50.36 Quite High
Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Taken together, these findings suggest that M3G may induce allodynia via activation of NO-ERK pathway, while maintenance of the allodynic response may be triggered by NO-activated astrocytes in the dorsal spinal cord.
M3G Spec (may) Positive_regulation (induce) of Positive_regulation (activation) of NO-ERK in spinal cord associated with allodynia and spinal cord
1) Confidence 0.14 Published 2009 Journal Int. Rev. Neurobiol. Section Abstract Doc Link 19607972 Disease Relevance 0.73 Pain Relevance 1.41

General Comments

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