INT161947

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Context Info
Confidence 0.52
First Reported 2008
Last Reported 2009
Negated 0
Speculated 0
Reported most in Title
Documents 2
Total Number 2
Disease Relevance 1.60
Pain Relevance 0.76

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (TGFBR1) aging (TGFBR1) cell motility (TGFBR1)
plasma membrane (TGFBR1) embryo development (TGFBR1)
Anatomy Link Frequency
stellate cells 1
TGFBR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Nerve growth factor 11 99.60 Very High Very High Very High
Pain 2 96.96 Very High Very High Very High
nociceptor 1 96.16 Very High Very High Very High
fibrosis 1 56.12 Quite High
COX2 1 5.00 Very Low Very Low Very Low
abdominal pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 2 96.96 Very High Very High Very High
Pancreatic Cancer 2 91.92 High High
Cirrhosis 3 90.92 High High
Cancer 31 89.60 High High
Apoptosis 6 88.96 High High
Primary Sclerosing Cholangitis 6 83.88 Quite High
Colorectal Cancer 50 60.56 Quite High
Familial Adenomatous Polyposis 48 44.84 Quite Low
Adenoma 33 33.84 Quite Low
Hyperplasia 15 33.12 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
receptor (TGFBR1) leading to activation of TGFBR1 protein kinase.
Positive_regulation (activation) of TGFBR1
1) Confidence 0.52 Published 2008 Journal Current Genomics Section Body Doc Link PMC2674304 Disease Relevance 0.60 Pain Relevance 0
Transforming growth factor-beta induces nerve growth factor expression in pancreatic stellate cells by activation of the ALK-5 pathway.
Positive_regulation (activation) of ALK-5 in stellate cells associated with nerve growth factor
2) Confidence 0.25 Published 2009 Journal Growth Factors Section Title Doc Link 19639490 Disease Relevance 0.99 Pain Relevance 0.76

General Comments

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