INT162842

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Context Info
Confidence 0.17
First Reported 2008
Last Reported 2009
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 2.48
Pain Relevance 0.96

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (CP) oxidoreductase activity (CP) extracellular region (CP)
transmembrane transport (CP)
Anatomy Link Frequency
edge 2
CP (Homo sapiens)
Pain Link Frequency Relevance Heat
Chronic pancreatitis 71 100.00 Very High Very High Very High
Pain 3 72.12 Quite High
alcohol 12 57.56 Quite High
fibrosis 12 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
abdominal pain 1 5.00 Very Low Very Low Very Low
medulla 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pancreatitis 95 100.00 Very High Very High Very High
Cerebral Palsy 10 100.00 Very High Very High Very High
Premature Aging 1 100.00 Very High Very High Very High
Aging 2 98.42 Very High Very High Very High
Injury 8 75.00 Quite High
Chronic Fatigue Syndrome 1 72.80 Quite High
Pain 1 72.12 Quite High
Fibrosis 11 5.00 Very Low Very Low Very Low
INFLAMMATION 5 5.00 Very Low Very Low Very Low
Primary Sclerosing Cholangitis 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We found that SPINK1 N34S is strongly associated with CP overall (OR 11.00; 95% CI: 7.59–15.93), but the effect of SPINK1 N34S in alcoholic CP (OR 4.98, 95% CI: 3.16–7.85) was significantly smaller than in idiopathic CP (OR 14.97, 95% C.I.?
Positive_regulation (overall) of Gene_expression (overall) of CP associated with chronic pancreatitis
1) Confidence 0.17 Published 2008 Journal PLoS ONE Section Abstract Doc Link PMC2289874 Disease Relevance 1.13 Pain Relevance 0.89
The goals of the workshop were to (1) define the current incidence and prevalence of CP, (2) review the known complications for persons aging with CP, (3) review current understanding of physiological processes that may contribute to loss of function and premature aging in CP, (4) evaluate current treatment interventions in terms of long-term outcomes, (5) identify cutting-edge technologies in neurorehabilitation that may help prevent or treat the effects of accelerated aging for persons diagnosed with CP, and (6) identify strategies to ensure that individuals with CP receive evidence-based care as they transition from pediatric to adult-care services.
Spec (may) Positive_regulation (contribute) of Gene_expression (aging) of CP in edge associated with premature aging, aging and cerebral palsy
2) Confidence 0.12 Published 2009 Journal Dev Med Child Neurol Section Abstract Doc Link 19740204 Disease Relevance 1.35 Pain Relevance 0.07

General Comments

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