INT164713

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Context Info
Confidence 0.32
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.43
Pain Relevance 0.69

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Acot1) lipid metabolic process (Acot1) cytoplasm (Acot1)
response to stress (Avp) signal transducer activity (Avp) cytosol (Acot1)
Anatomy Link Frequency
CdN 3
Avp (Rattus norvegicus)
Acot1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antagonist 3 99.96 Very High Very High Very High
Pain 6 98.24 Very High Very High Very High
Analgesic 1 81.28 Quite High
Neurotransmitter 1 78.88 Quite High
Disease Link Frequency Relevance Heat
Pain 5 98.24 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results showed that pain stimulation increased both AVP and Ach concentrations in the CdN perfusion liquid; AVP increased Ach concentration in the CdN perfusion liquid, while AVP receptor antagonist including d(CH(2))(5)Tyr(Me)AVP (V(1) receptor antagonist) and d(CH(2))(5)[D-Ile(2), Ile(4), Ala-NH(2)(9)]AVP (V(2) receptor antagonist) decreased Ach concentration in the CdN perfusion liquid.
AVP Binding (receptor) of in CdN Positive_regulation (increased) of Localization (concentration) of Ach in CdN associated with pain and antagonist
1) Confidence 0.32 Published 2010 Journal Peptides Section Abstract Doc Link 19948196 Disease Relevance 0.43 Pain Relevance 0.69

General Comments

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