INT164926

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Context Info
Confidence 0.46
First Reported 2007
Last Reported 2010
Negated 2
Speculated 3
Reported most in Body
Documents 9
Total Number 14
Disease Relevance 16.66
Pain Relevance 2.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (GYPC)
Anatomy Link Frequency
plasma 1
GYPC (Homo sapiens)
Pain Link Frequency Relevance Heat
Cholecystokinin 279 99.00 Very High Very High Very High
antagonist 21 97.08 Very High Very High Very High
Paracetamol 17 93.76 High High
nud 4 83.84 Quite High
spastic colon 3 81.44 Quite High
Opioid 27 80.48 Quite High
cytokine 32 64.92 Quite High
analgesia 8 62.92 Quite High
Pain 8 58.84 Quite High
Inflammatory mediators 16 52.16 Quite High
Disease Link Frequency Relevance Heat
Gastric Motility Disorder 840 100.00 Very High Very High Very High
Critical Illness 426 99.84 Very High Very High Very High
Disease 8 97.92 Very High Very High Very High
Liver Disease 16 97.24 Very High Very High Very High
Diabetes Mellitus 75 97.04 Very High Very High Very High
Chronic Renal Failure 16 94.16 High High
Sleep Disorders 20 91.32 High High
Burns 88 91.28 High High
Myocardial Infarction 11 91.24 High High
Natriuresis 8 89.44 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
After other factors that influence GE were controlled for, there was a modest effect of admission diagnosis on GE (r = 0.48; P = 0.02) with linear and hierarchical regression analyses.
Regulation (influence) of GE associated with gastric motility disorder
1) Confidence 0.46 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 2.17 Pain Relevance 0.08
The 13CO2 concentration over time was plotted and the resultant curves were used to calculate a GE coefficient (GEC), a global index for the GE rate, which accounts for the rates of both appearance and disappearance of the label in breath [20].
Regulation (calculate) of GE associated with gastric motility disorder
2) Confidence 0.33 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 0.48 Pain Relevance 0
In addition, this technique has not been validated against scintigraphy for measurement of GE in critically ill patients.
Neg (not) Regulation (validated) of GE associated with critical illness and gastric motility disorder
3) Confidence 0.33 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 1.67 Pain Relevance 0.21
Our findings show that admission diagnosis has a significant but modest impact on GE in critically ill patients after controlling for other variables that may influence GE.
Spec (may) Regulation (influence) of GE associated with critical illness and gastric motility disorder
4) Confidence 0.20 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 2.09 Pain Relevance 0.05
These findings suggest that critical illness may have an 'additive' adverse effect on the disturbed GE in these patients.
Regulation (effect) of GE associated with critical illness and gastric motility disorder
5) Confidence 0.20 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 2.48 Pain Relevance 0.17
In the current study, pre-existing co-morbidities had a moderate effect on GE during critical illness.
Regulation (effect) of GE associated with critical illness and gastric motility disorder
6) Confidence 0.20 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 2.54 Pain Relevance 0.32
After other factors that influence GE were controlled for, there was a modest effect of admission diagnosis on GE (r = 0.48; P = 0.02) with linear and hierarchical regression analyses.
Spec (analyses) Regulation (effect) of GE associated with gastric motility disorder
7) Confidence 0.20 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 2.21 Pain Relevance 0
However, because the association remained at day eight with other factors known to alter GE in critical illness having been controlled for, the relationship between admission diagnosis and GE may have been even stronger if GE had been assessed within the first three days of admission.
Regulation (alter) of GE associated with critical illness and gastric motility disorder
8) Confidence 0.20 Published 2007 Journal Crit Care Section Body Doc Link PMC2151889 Disease Relevance 1.30 Pain Relevance 0
We investigated the role of glucose-dependent insulintropic polypeptide (GIP) in the regulation of gastric emptying (GE), appetite, energy intake (EI), energy expenditure (EE), plasma levels of triglycerides (TAG), and free fatty acids (FFA) in humans.
Regulation (regulation) of GE in plasma
9) Confidence 0.10 Published 2010 Journal Am. J. Physiol. Endocrinol. Metab. Section Abstract Doc Link 19996386 Disease Relevance 0 Pain Relevance 0.08
Whilst the present observations strengthen the rationale for the potential use of CCK antagonists in the management of feed intolerance in the critically ill, it should be recognised that the efficacy of such agents may be limited due to the complex interaction amongst many factors that regulate GE during critical illness.
Regulation (regulate) of GE associated with critical illness, antagonist and cholecystokinin
10) Confidence 0.06 Published 2007 Journal Crit Care Section Body Doc Link PMC2246231 Disease Relevance 0.50 Pain Relevance 0.54
The effects of PYY antagonism on GE in humans, however, are unknown.
Regulation (effects) of GE
11) Confidence 0.04 Published 2007 Journal Crit Care Section Body Doc Link PMC2246231 Disease Relevance 0.31 Pain Relevance 0.40
The observation that the rate of GE is inversely related to the fasting levels of CCK and PYY suggests that they may contribute to the regulation of GE in critically ill patients.
Regulation (regulation) of GE associated with critical illness and cholecystokinin
12) Confidence 0.02 Published 2007 Journal Crit Care Section Body Doc Link PMC2246231 Disease Relevance 0.67 Pain Relevance 0.18
In the present study we sought to determine the effect of heat on GE.
Spec (determine) Regulation (effect) of GE
13) Confidence 0.00 Published 2007 Journal Eur J Appl Physiol Section Body Doc Link PMC2039810 Disease Relevance 0.24 Pain Relevance 0
No significant change in GE within trials was observed comparing GE over the first half (min 5–8) with GE over the second half (min 15–18).Fig. 1Gross-efficiency (GE) plotted over time at 60% in the thermo-neutral (N) climate (dashed line) and in the hot dry (H) climate (solid line) ±SD (dotted lines)
Neg (No) Regulation (change) of GE
14) Confidence 0.00 Published 2007 Journal Eur J Appl Physiol Section Body Doc Link PMC2039810 Disease Relevance 0 Pain Relevance 0

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