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Context Info
Confidence 0.43
First Reported 2009
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.61
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (LCN2)
LCN2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Angina 1 50.00 Quite Low
Inflammation 3 45.76 Quite Low
fibrosis 2 38.00 Quite Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
lidocaine 1 5.00 Very Low Very Low Very Low
Inflammatory mediators 1 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
rheumatoid arthritis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 4 85.96 High High
Renal Disease 1 81.04 Quite High
Emphysema 3 74.00 Quite High
Stress 8 60.64 Quite High
Pulmonary Emphysema 1 59.72 Quite High
Lung Injury 1 55.52 Quite High
Chronic Renal Failure 1 50.00 Quite Low
Cv General 3 Under Development 1 50.00 Quite Low
INFLAMMATION 6 45.76 Quite Low
Cystic Fibrosis 1 38.00 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore lack of NGAL protein is one important reason of remaining and progressive lesion.
Negative_regulation (lack) of NGAL
1) Confidence 0.43 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC2873661 Disease Relevance 0.36 Pain Relevance 0
RESULTS: In both groups we found a significant rise in serum NGAL after 2, 4, and 8 hours, and in urinary NGAL and IL-18 after 4, 8, and 24 hours after cardiac catheterization.
Negative_regulation (rise) of NGAL
2) Confidence 0.05 Published 2009 Journal Ren Fail Section Body Doc Link 20030526 Disease Relevance 0.25 Pain Relevance 0

General Comments

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