INT165328

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Context Info
Confidence 0.21
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.09
Pain Relevance 0.31

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Nkx6-2) nucleus (Nkx6-2) DNA binding (Nkx6-2)
Anatomy Link Frequency
autonomic 1
Nkx6-2 (Mus musculus)
Pain Link Frequency Relevance Heat
tetrodotoxin 1 96.76 Very High Very High Very High
gABA 3 90.88 High High
Glutamate 3 90.52 High High
Disease Link Frequency Relevance Heat
Poisoning 2 87.16 High High
Heart Rate Under Development 1 42.56 Quite Low
Vomiting 1 41.88 Quite Low
Sialorrhea 1 41.48 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The present study indicates that GTX increases Ca(2+) influx through voltage-dependent Ca(2+) channels secondary to activation of voltage-dependent Na(+) channels in inhibitory and excitatory nerve terminals synapsing on VMH neurons, and the subsequent increased release of GABA and glutamate from these terminals may be responsible for the autonomic symptoms of GTX intoxication.
Positive_regulation (increases) of GTX in autonomic associated with glutamate, gaba and poisoning
1) Confidence 0.21 Published 2010 Journal Neurotoxicology Section Abstract Doc Link 20038438 Disease Relevance 0.09 Pain Relevance 0.31

General Comments

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