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Context Info
Confidence 0.02
First Reported 2006
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.27
Pain Relevance 0.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (ABCA1) small molecule metabolic process (ABCA1) Golgi apparatus (ABCA1)
ATPase activity (ABCA1) plasma membrane (ABCA1) intracellular (ABCA1)
Anatomy Link Frequency
cleavage 2
ABCA1 (Homo sapiens)
Pain Link Frequency Relevance Heat
chemokine 1 99.96 Very High Very High Very High
Neuropathic pain 1 99.92 Very High Very High Very High
diclofenac 12 96.14 Very High Very High Very High
Paracetamol 7 95.66 Very High Very High Very High
metalloproteinase 2 79.72 Quite High
carbamazepine 8 78.88 Quite High
fibrosis 4 30.08 Quite Low
Inflammation 55 22.96 Low Low
Abeta 12 5.00 Very Low Very Low Very Low
cytokine 11 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neuropathic Pain 1 99.92 Very High Very High Very High
Disease 8 89.44 High High
Apoptosis 14 76.32 Quite High
Cancer 31 50.00 Quite Low
Pulmonary Fibrosis 4 30.08 Quite Low
Lung Cancer 7 28.72 Quite Low
Diabetes Mellitus 13 26.68 Quite Low
Increased Venous Pressure Under Development 4 25.08 Quite Low
INFLAMMATION 63 22.96 Low Low
Stress 13 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Several pharmaceutical products (e.g. ibuprofen, naproxen, acetaminophen, ketoprofen, diclofenac, bezafibrate, gemfibrozil, ranitidine, ofloxacin, hydrochlorothiazide, and paroxetine) with high rates of attenuation can be expected to be completely removed from wastewater by adsorption or degradation, or a combination of both, during membrane treatment.
Protein_catabolism (degradation) of membrane associated with paracetamol and diclofenac
1) Confidence 0.02 Published 2006 Journal Anal Bioanal Chem Section Body Doc Link PMC1805043 Disease Relevance 0 Pain Relevance 0.18
This cleavage process is modulated by Ca++ levels, and following proteolytic cleavage the remaining membrane-bound C-terminal fragment is subject to further degradation by ?
Protein_catabolism (degradation) of membrane-bound in cleavage
2) Confidence 0.02 Published 2009 Journal J Transl Med Section Body Doc Link PMC2666642 Disease Relevance 0.08 Pain Relevance 0.04
Extracellular cathepsin S may also be involved in angiogenesis and initiation and/or maintenance of neuropathic pain by cleavage of the membrane-bound chemokine fractalkine (CX3CL1).
Protein_catabolism (cleavage) of membrane-bound in cleavage associated with chemokine and neuropathic pain
3) Confidence 0.01 Published 2010 Journal Curr Top Med Chem Section Abstract Doc Link 20337580 Disease Relevance 0.19 Pain Relevance 0.15

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