INT168225

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Context Info
Confidence 0.12
First Reported 2006
Last Reported 2010
Negated 1
Speculated 1
Reported most in Body
Documents 3
Total Number 4
Disease Relevance 0.09
Pain Relevance 0.50

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
decidua 1
cortex 1
Upper 1
ge (Mus musculus)
Pain Link Frequency Relevance Heat
gABA 10 99.32 Very High Very High Very High
GABAergic 23 96.88 Very High Very High Very High
agonist 22 86.16 High High
Dopamine 1 51.28 Quite High
abdominal pain 4 50.00 Quite Low
Enkephalin 1 49.60 Quite Low
interneuron 5 5.00 Very Low Very Low Very Low
anesthesia 4 5.00 Very Low Very Low Very Low
Thalamus 4 5.00 Very Low Very Low Very Low
Hippocampus 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Abdominal Pain 4 94.92 High High
Gastric Motility Disorder 4 50.00 Quite Low
Targeted Disruption 23 5.00 Very Low Very Low Very Low
Epilepsy 14 5.00 Very Low Very Low Very Low
Intellectual Impairment 14 5.00 Very Low Very Low Very Low
Lissencephaly 10 5.00 Very Low Very Low Very Low
Syndrome 10 5.00 Very Low Very Low Very Low
Congenital Anomalies 9 5.00 Very Low Very Low Very Low
Convulsion 8 5.00 Very Low Very Low Very Low
Infantile Spasms 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
METHODS: Patients presumed to have GP underwent 4-hour GE scintigraphy and upper endoscopy examinations and completed the following: patient assessments of gastrointestinal symptoms (Patient Assessment of Upper Gastrointestinal Symptom Severity Index), abdominal pain questionnaires (Short-Form of the McGill Pain Questionnaire), and quality-of-life questionnaires.
Spec (examinations) Gene_expression (scintigraphy) of GE in Upper
1) Confidence 0.12 Published 2010 Journal Clin. Gastroenterol. Hepatol. Section Body Doc Link 20472097 Disease Relevance 0.09 Pain Relevance 0
Moreover, ARX overexpression in cortical progenitors in vivo or in dissociated cultures from E16 rat cortex or GE does not induce GABA or calbindin expression, suggesting that even if ARX is involved in GABAergic specification, it does not appear to be sufficient by itself to induce the GABAergic phenotype and thus, it may act in combination with other genes (Friocourt et al., 2008).
Gene_expression (overexpression) of GE in cortex associated with gaba and gabaergic
2) Confidence 0.04 Published 2010 Journal Frontiers in Cellular Neuroscience Section Body Doc Link PMC2841486 Disease Relevance 0 Pain Relevance 0.46
None of the treatment groups were different from OvxC in PR expression of the GE (data not shown).
Gene_expression (expression) of GE
3) Confidence 0.01 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1586009 Disease Relevance 0 Pain Relevance 0
In early pregnancy the LE (Fig 4F) and GE were negative, while the decidua was faintly stained (Table 3).
Neg (negative) Gene_expression (negative) of GE in decidua
4) Confidence 0.01 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1586009 Disease Relevance 0 Pain Relevance 0.04

General Comments

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