INT169286

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Context Info
Confidence 0.45
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.18
Pain Relevance 0.57

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

response to stress (CNR1, EGFR) signal transducer activity (CNR1, EGFR) plasma membrane (CNR1, EGFR)
endosome (EGFR) cell morphogenesis (EGFR) aging (CNR1)
CNR1 (Homo sapiens)
EGFR (Homo sapiens)
Pain Link Frequency Relevance Heat
Cannabinoid receptor 10 100.00 Very High Very High Very High
agonist 2 100.00 Very High Very High Very High
qutenza 2 100.00 Very High Very High Very High
antagonist 2 74.96 Quite High
Disease Link Frequency Relevance Heat
Wound Healing 1 97.52 Very High Very High Very High
Injury 1 87.04 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Both the selective CB1 and TRPV1 agonists (i.e., WIN55,212-2 [WIN] and capsaicin [CAP], respectively) induced EGFR phosphorylation whereas either inhibition of its tyrosine kinase activity with AG1478 or functional blockage eliminated this response.
CB1 Positive_regulation (induced) of Phosphorylation (phosphorylation) of EGFR associated with qutenza, cannabinoid receptor and agonist
1) Confidence 0.45 Published 2010 Journal Exp. Eye Res. Section Abstract Doc Link 20619260 Disease Relevance 0.18 Pain Relevance 0.57

General Comments

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