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Context Info
Confidence 0.21
First Reported 1991
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 0.56
Pain Relevance 0.53

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (AKR1B10) cellular_component (AKR1B10) lysosome (AKR1B10)
Anatomy Link Frequency
ventral 1
AKR1B10 (Homo sapiens)
Pain Link Frequency Relevance Heat
spinothalamic 7 98.88 Very High Very High Very High
Enkephalin 2 80.56 Quite High
Somatosensory cortex 1 78.00 Quite High
primary somatosensory cortex 4 75.00 Quite High
nociceptor 1 25.00 Low Low
Pain 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 5 91.20 High High
Nociception 3 80.88 Quite High
Stress 3 75.40 Quite High
Myocardial Infarction 23 38.44 Quite Low
Critical Illness 2 26.40 Quite Low
Pain 2 5.00 Very Low Very Low Very Low
Coma 2 5.00 Very Low Very Low Very Low
Sepsis 1 5.00 Very Low Very Low Very Low
Chronic Disease 1 5.00 Very Low Very Low Very Low
Mental Disorders 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
What is the quality of life or HSI of such a patient?
Gene_expression (is) of HSI
1) Confidence 0.21 Published 2008 Journal Crit Care Section Body Doc Link PMC2575575 Disease Relevance 0.25 Pain Relevance 0
Quantitative analysis of the positions and numbers of retrogradely labeled neurons and anterogradely labeled terminal fields reveal that: 1) ventral posterior lateral (VPL), ventral posterior inferior (VPI), and central lateral (CL), combined, receive 87% of spinothalamic inputs originating from the cervical enlargement; 2) these three nuclei contain over 91% of all thalamocortical neurons projecting to hSI that are likely to receive STT input; and 3) these putatively contacted neurons account for less than 24% of all thalamic projections to hSI.
Gene_expression (projecting) of hSI in ventral associated with spinothalamic
2) Confidence 0.04 Published 1991 Journal J. Comp. Neurol. Section Abstract Doc Link 1865012 Disease Relevance 0.16 Pain Relevance 0.45
In situ hybridization revealed that novelty or HSI elevated levels of PPE hnRNA and c-fos mRNA in the PVN.
Gene_expression (levels) of HSI in PVN
3) Confidence 0.03 Published 1997 Journal Brain Res. Section Abstract Doc Link 9295199 Disease Relevance 0.15 Pain Relevance 0.08

General Comments

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