INT169864

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.45
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.38
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transmembrane transport (SCN2A)
Anatomy Link Frequency
neurons 2
cerebellum 2
SCN2A (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 6 50.00 Quite Low
sodium channel 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Epilepsy 4 69.40 Quite High
Febrile Convulsions 2 68.16 Quite High
Convulsion 2 67.44 Quite High
Pain 2 50.00 Quite Low
Myoclonus 2 50.00 Quite Low
Ataxia 2 50.00 Quite Low
Channelopathies 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Immunohistochemical studies suggest a developmentally increasing expression of Na(V)1.2 in granule cell axons projecting to Purkinje neurons.
Positive_regulation (increasing) of Gene_expression (expression) of Na(V)1.2 in neurons
1) Confidence 0.45 Published 2010 Journal Neurology Section Body Doc Link 20956790 Disease Relevance 0.20 Pain Relevance 0
The developmentally increasing expression of Na(V)1.2 in cerebellum may be responsible for the later onset of episodic ataxia.


Positive_regulation (increasing) of Gene_expression (expression) of Na(V)1.2 in cerebellum
2) Confidence 0.45 Published 2010 Journal Neurology Section Body Doc Link 20956790 Disease Relevance 0.17 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox