INT170163

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Context Info
Confidence 0.16
First Reported 2001
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 3.56
Pain Relevance 0.96

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (ACSM3) molecular_function (ACSM3) cellular_component (ACSM3)
ligase activity (ACSM3) lipid metabolic process (ACSM3)
Anatomy Link Frequency
plasma 1
ACSM3 (Homo sapiens)
Pain Link Frequency Relevance Heat
cva 103 100.00 Very High Very High Very High
Catechol-O-methyltransferase 109 99.84 Very High Very High Very High
Glutamate 1 88.88 High High
midbrain 1 72.52 Quite High
Thalamus 1 71.40 Quite High
imagery 16 60.32 Quite High
Calcium channel 2 57.92 Quite High
Catecholamine 7 33.04 Quite Low
Inflammation 1 28.80 Quite Low
Pain 21 21.88 Low Low
Disease Link Frequency Relevance Heat
Cv General 4 Under Development 54 100.00 Very High Very High Very High
Subarachnoid Hemorrhage 6 100.00 Very High Very High Very High
Disease 31 99.56 Very High Very High Very High
Cancer 2 96.56 Very High Very High Very High
Cardiovascular Disease 2 95.68 Very High Very High Very High
Neurodegenerative Disease 2 94.72 High High
Muscular Spasm 6 94.44 High High
Cv General 3 Under Development 46 93.28 High High
Rupture 20 93.16 High High
Hyperhomocysteinemia 2 89.56 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The efficacy of endovascular treatment in the prevention of SAH remains unknown [16,17].
Negative_regulation (prevention) of SAH associated with cva
1) Confidence 0.16 Published 2008 Journal Trials Section Body Doc Link PMC2526062 Disease Relevance 1.19 Pain Relevance 0.19
Homocysteine is a precursor for the biosynthesis of S-adenosyl-L-homocysteine (SAH) which is a strong, noncompetitive inhibitor of COMT.
Negative_regulation (inhibitor) of SAH associated with catechol-o-methyltransferase
2) Confidence 0.14 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2664927 Disease Relevance 0.85 Pain Relevance 0.29
The relatively high prevalence of IAs and aneurysmal SAH place the disease in the group of moderately common disorders.
Negative_regulation (prevalence) of SAH associated with cva and disease
3) Confidence 0.09 Published 2002 Journal BMC Med Genet Section Body Doc Link PMC119849 Disease Relevance 0.26 Pain Relevance 0.05
Since autoregulation is impaired after SAH (Tenjin et al 1988), CBF becomes totally dependent on the CPP.
Negative_regulation (impaired) of SAH associated with cva
4) Confidence 0.07 Published 2007 Journal Vascular Health and Risk Management Section Body Doc Link PMC2350130 Disease Relevance 0.63 Pain Relevance 0.24
From our findings, we can say that CNP concentration in the CSF was high in the acute phase and decreased gradually after SAH, whereas CNP in the plasma did not change.
Negative_regulation (decreased) of SAH in plasma associated with cva
5) Confidence 0.03 Published 2001 Journal Crit Care Section Body Doc Link PMC29055 Disease Relevance 0.63 Pain Relevance 0.19

General Comments

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