INT170183

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Context Info
Confidence 0.26
First Reported 2001
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 1.90
Pain Relevance 0.57

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (SULT2A1) small molecule metabolic process (SULT2A1) cytoplasm (SULT2A1)
Anatomy Link Frequency
liver 2
SULT2A1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 14 99.44 Very High Very High Very High
Arthritis 45 94.72 High High
Inflammation 18 84.16 Quite High
rheumatoid arthritis 34 81.28 Quite High
dexamethasone 3 80.52 Quite High
agonist 39 75.12 Quite High
Bile 27 50.00 Quite Low
Kinase C 12 5.00 Very Low Very Low Very Low
Paracetamol 7 5.00 Very Low Very Low Very Low
depression 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 27 99.14 Very High Very High Very High
Chronic Fatigue Syndrome 52 97.90 Very High Very High Very High
Arthritis 43 94.72 High High
Targeted Disruption 6 86.64 High High
INFLAMMATION 12 84.16 Quite High
Rheumatoid Arthritis 44 81.28 Quite High
Frailty 1 78.76 Quite High
Fatigue 8 75.28 Quite High
Van Bogaert's Disease 21 69.08 Quite High
Obesity 9 58.44 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Here, we do not attempt to be comprehensive but present several findings related to the regulation of hepatic SULT2A expression as an example.
Regulation (regulation) of Gene_expression (expression) of SULT2A
1) Confidence 0.26 Published 2009 Journal PPAR Research Section Body Doc Link PMC2724710 Disease Relevance 0.06 Pain Relevance 0.07
-inducible human hepatic SULT2A1 expression occurs through a distal PPRE in the 5?
Regulation (through) of Gene_expression (expression) of SULT2A1
2) Confidence 0.26 Published 2009 Journal PPAR Research Section Body Doc Link PMC2724710 Disease Relevance 0.26 Pain Relevance 0.04
After we had controlled for the ESR, previous glucocorticoid usage, duration of disease, current NSAID treatment, and HOMAIR, the differences in DHEAS concentrations between patients and controls were markedly attenuated in women (P = 0.050) and were no longer present in men (P = 0.133).
Regulation (differences) of Gene_expression (concentrations) of DHEAS associated with cinod and disease
3) Confidence 0.26 Published 2001 Journal Arthritis Res Section Body Doc Link PMC30711 Disease Relevance 0.94 Pain Relevance 0.43
Cleare et al.36 found higher basal DHEA levels in CFS patients with no psychiatric comorbidity compared to healthy controls; however, they did not report any difference in DHEAS levels.
Regulation (difference) of Gene_expression (levels) of DHEAS associated with chronic fatigue syndrome
4) Confidence 0.22 Published 2009 Journal Psychiatry Investigation Section Body Doc Link PMC2796068 Disease Relevance 0.64 Pain Relevance 0
However, unlike rodent hepatic SULT2As, treatment of human liver cells with rifampicin, the prototypical ligand activator of human PXR [130], produces a biphasic effect on SULT2A1 expression [131].
Regulation (effect) of Gene_expression (expression) of SULT2A1 in liver
5) Confidence 0.16 Published 2009 Journal PPAR Research Section Body Doc Link PMC2724710 Disease Relevance 0 Pain Relevance 0.04

General Comments

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