INT170694

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Context Info
Confidence 0.35
First Reported 2001
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0
Pain Relevance 0.43

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Calm2) nucleoplasm (Calm2) cytoskeleton (Calm2)
cell cycle (Calm2) cellular_component (Calm2) biological_process (Calm2)
Anatomy Link Frequency
plasma 1
Calm2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 78 100.00 Very High Very High Very High
agonist 21 80.04 Quite High
Glutamate 18 68.92 Quite High
Glutamate receptor 7 50.00 Quite Low
tetrodotoxin 20 12.48 Low Low
addiction 18 12.32 Low Low
imagery 9 5.00 Very Low Very Low Very Low
Action potential 5 5.00 Very Low Very Low Very Low
nMDA receptor 4 5.00 Very Low Very Low Very Low
antagonist 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 20 24.76 Low Low
Nash(non-alcoholic Steatohepatitis) 12 5.00 Very Low Very Low Very Low
Myelodysplastic Syndromes 3 5.00 Very Low Very Low Very Low
Infection 3 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low
Apoptosis 1 5.00 Very Low Very Low Very Low
Cognitive Disorder 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Aging 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Phosphorylation of the receptors at specific sites prevents CaM binding to mGluRs [25].
Phosphorylation (Phosphorylation) of CaM
1) Confidence 0.35 Published 2001 Journal BMC Neurosci Section Body Doc Link PMC58838 Disease Relevance 0 Pain Relevance 0.07
MARCKS is normally associated with the plasma membrane and translocates to the cytoplasm on phosphorylation by PKC and/or CaM binding (Graff et al. 1989; Arbuzova et al. 2002).
Phosphorylation (phosphorylation) of CaM in plasma associated with kinase c
2) Confidence 0.13 Published 2007 Journal Journal of Neurochemistry Section Body Doc Link PMC2658029 Disease Relevance 0 Pain Relevance 0.37
However, whether L-type channels enlist this resident CaM to signal to CREB after brief depolarization is unclear, as are the nature of rapid coupling between depolarization and CREB phosphorylation and the identity of downstream molecular mechanisms.
Phosphorylation (phosphorylation) of CaM
3) Confidence 0.07 Published 2008 Journal The Journal of Cell Biology Section Body Doc Link PMC2592819 Disease Relevance 0 Pain Relevance 0

General Comments

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