INT170700

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Context Info
Confidence 0.50
First Reported 2001
Last Reported 2001
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 0.36
Pain Relevance 0.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Siah1a) cytosol (Siah1a) nucleus (Siah1a)
cell cycle (Siah1a) ligase activity (Siah1a) cytoplasm (Siah1a)
Siah1a (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Glutamate 36 77.52 Quite High
addiction 3 59.04 Quite High
Glutamate receptor 12 50.00 Quite Low
imagery 6 5.00 Very Low Very Low Very Low
tetrodotoxin 3 5.00 Very Low Very Low Very Low
Neuronal excitability 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 18 87.92 High High
Apoptosis 3 53.80 Quite High
Cancer 3 53.16 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Next, when CaM was coexpressed with mGluR5b and Siah1a, calcium current modulation was significantly increased (over that in cells expressing mGluR5b and Siah1a) to 25 ± 5% (n = 12; figure 6).
Positive_regulation (increased) of Gene_expression (coexpressed) of Siah1a
1) Confidence 0.50 Published 2001 Journal BMC Neurosci Section Body Doc Link PMC58838 Disease Relevance 0 Pain Relevance 0
Finally, the effect of CaM overexpression on Siah1a function was assessed.


Positive_regulation (overexpression) of Gene_expression (overexpression) of Siah1a
2) Confidence 0.50 Published 2001 Journal BMC Neurosci Section Body Doc Link PMC58838 Disease Relevance 0.36 Pain Relevance 0.06
The reduction in group I mGluR signaling induced by Siah1a expression may be attributed to several possible mechanisms.
Positive_regulation (induced) of Gene_expression (expression) of Siah1a
3) Confidence 0.50 Published 2001 Journal BMC Neurosci Section Body Doc Link PMC58838 Disease Relevance 0 Pain Relevance 0.06

General Comments

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