INT171008

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Context Info
Confidence 0.15
First Reported 2002
Last Reported 2002
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.33
Pain Relevance 0.89

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (IL10) extracellular region (IL10) intracellular (IL10)
cell-cell signaling (IL10) response to stress (IL10)
IL10 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 91 100.00 Very High Very High Very High
corticosteroid 1 99.18 Very High Very High Very High
antagonist 7 99.00 Very High Very High Very High
cINOD 3 96.28 Very High Very High Very High
chemokine 29 90.28 High High
cytokine 44 88.76 High High
dexamethasone 7 81.84 Quite High
metalloproteinase 3 44.16 Quite Low
Inflammatory mediators 10 5.00 Very Low Very Low Very Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 111 100.00 Very High Very High Very High
Adhesions 9 94.28 High High
Asthma 41 75.76 Quite High
Airway Remodeling 13 51.12 Quite High
Disease 5 46.40 Quite Low
Wound Healing 1 31.60 Quite Low
Hyperplasia 4 13.76 Low Low
Hypertrophy 4 5.00 Very Low Very Low Very Low
Fibrosis 3 5.00 Very Low Very Low Very Low
Occupational Lung Diseases 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Corticosteroids may also control airway inflammation by increasing the transcription of anti-inflammatory genes like IL-10, IL-12 or IL-1 receptor antagonist, the gene products of which appear to be the most potent anti-inflammatory drugs for use in the treatment of airway inflammation [63].
Positive_regulation (increasing) of Transcription (transcription) of IL-10 associated with corticosteroid, inflammation, antagonist and cinod
1) Confidence 0.15 Published 2002 Journal Respir Res Section Body Doc Link PMC64809 Disease Relevance 1.33 Pain Relevance 0.89

General Comments

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