INT171677

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Context Info
Confidence 0.00
First Reported 2002
Last Reported 2002
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.33

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (PRKCA, CISD1) nucleoplasm (PRKCA) small molecule metabolic process (PRKCA)
endoplasmic reticulum (PRKCA) enzyme binding (PRKCA) cytoplasm (PRKCA)
PRKCA (Homo sapiens)
CISD1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Kinase C 40 100.00 Very High Very High Very High
antagonist 4 52.20 Quite High
Pain 2 49.60 Quite Low
adenocard 18 5.00 Very Low Very Low Very Low
ischemia 13 5.00 Very Low Very Low Very Low
agonist 4 5.00 Very Low Very Low Very Low
Opioid 2 5.00 Very Low Very Low Very Low
analgesia 1 5.00 Very Low Very Low Very Low
beta blocker 1 5.00 Very Low Very Low Very Low
potassium channel 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 2 49.60 Quite Low
Shock 2 9.60 Low Low
Cv Unclassified Under Development 13 5.00 Very Low Very Low Very Low
Injury 7 5.00 Very Low Very Low Very Low
Necrosis 2 5.00 Very Low Very Low Very Low
Diabetes Mellitus 1 5.00 Very Low Very Low Very Low
Hypertension 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low
Stress 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
There is evidence that mitoKATP channel opening increases radical oxygen species production which in turn may activate PKC, [18] but it is well known that mitoKATP channels are also modulated by PKC [19].
PKC Regulation (modulated) of mitoKATP associated with kinase c
1) Confidence 0.00 Published 2002 Journal BMC Physiol Section Body Doc Link PMC117790 Disease Relevance 0 Pain Relevance 0.33

General Comments

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