INT172189

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Context Info
Confidence 0.30
First Reported 2002
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 6
Total Number 6
Disease Relevance 1.81
Pain Relevance 0.29

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

CHR (Homo sapiens)
Pain Link Frequency Relevance Heat
Calcium channel 52 81.28 Quite High
potassium channel 1 70.72 Quite High
Neuropathic pain 1 58.08 Quite High
cytokine 40 38.72 Quite Low
Inflammatory stimuli 6 19.28 Low Low
Inflammatory response 6 15.84 Low Low
Inflammation 35 5.00 Very Low Very Low Very Low
Catecholamine 18 5.00 Very Low Very Low Very Low
cocaine 17 5.00 Very Low Very Low Very Low
withdrawal 17 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Carcinoma 95 100.00 Very High Very High Very High
Infection 61 89.44 High High
Adhesions 9 88.12 High High
Sepsis 7 86.88 High High
Sprains And Strains 165 83.08 Quite High
Death 6 82.32 Quite High
Renal Cancer 18 80.40 Quite High
Cancer 45 71.28 Quite High
Neuropathic Pain 1 58.08 Quite High
Fungal Infection 3 26.88 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Nor did the result for KRT7 (cytokeratin 7), which has been shown to be overexpressed in Chr [20].
Gene_expression (overexpressed) of Chr associated with carcinoma
1) Confidence 0.30 Published 2002 Journal BMC Bioinformatics Section Body Doc Link PMC130042 Disease Relevance 1.07 Pain Relevance 0
The present experiments have demonstrated that the two CgA peptides, CHR and CAT, penetrate rapidly into PMNs (Figure 5A) and subsequently interact with CaM (Figure 5C) to activate iPLA2 (Figure 6C).
Gene_expression (peptides) of CHR
2) Confidence 0.16 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2639705 Disease Relevance 0.14 Pain Relevance 0.04
In contrast, the control peptide, rhodamine-labeled Hippocampal Cholinergic Neurostimulating Peptide (HCNP), unable to induce Ca2+ influx into PMNs (not shown), was not detected in the cytoplasm, indicating a specificity for the entry of rhodamine-labeled CHR and CAT (Figure 5A).
Gene_expression (labeled) of CHR
3) Confidence 0.16 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2639705 Disease Relevance 0 Pain Relevance 0.04
An immediate increase of activated PMNs (M1/M2) was observed 15 s after the application of either CHR or CAT (Figure 2A).
Gene_expression (application) of CHR
4) Confidence 0.14 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2639705 Disease Relevance 0 Pain Relevance 0.11
In females, suggestive QTL were detected on Chr 1 and 9 for Taylor I (Fig. 2a), and Chr 8 for the recent RI set (Fig. 2b).
Gene_expression (detected) of Chr
5) Confidence 0.01 Published 2010 Journal Genes, Brain, and Behavior Section Body Doc Link PMC2855868 Disease Relevance 0.11 Pain Relevance 0.09
First, we used whole genome transcription profiling to identify 191 (56 on chr. 8, 100 on chr. 11, and 35 on chr. 18) genes on our three chromosomes of interest that are differentially expressed between S. aureus-infected A/J and C57BL/6J.
Gene_expression (expressed) of chr
6) Confidence 0.01 Published 2010 Journal PLoS Pathogens Section Abstract Doc Link PMC2932726 Disease Relevance 0.49 Pain Relevance 0

General Comments

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