INT172906

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Context Info
Confidence 0.16
First Reported 2003
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.03
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Bax, Bcl2l1) intracellular (Bax, Bcl2l1) cytoplasm (Bax, Bcl2l1)
cytosol (Bax, Bcl2l1) cell proliferation (Bax, Bcl2l1) endoplasmic reticulum (Bax)
Bax (Mus musculus)
Bcl2l1 (Mus musculus)
Pain Link Frequency Relevance Heat
metalloproteinase 1 42.24 Quite Low
cINOD 18 36.08 Quite Low
COX-2 inhibitor 10 12.16 Low Low
aspirin 8 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
antagonist 3 5.00 Very Low Very Low Very Low
Paracetamol 1 5.00 Very Low Very Low Very Low
Analgesic 1 5.00 Very Low Very Low Very Low
headache 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 5 99.20 Very High Very High Very High
Apoptosis 73 99.08 Very High Very High Very High
Pancreatic Cancer 84 75.12 Quite High
Cancer 95 64.00 Quite High
Adhesions 3 61.76 Quite High
Breast Cancer 2 58.76 Quite High
Hepatocellular Cancer 16 33.92 Quite Low
Repression 1 16.56 Low Low
Carcinoma 2 16.24 Low Low
Adenocarcinoma 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In particular, Bax can homodimerize with itself and heterodimerize with Bcl-2 or Bcl-xL.
Bax Binding (heterodimerize) of Bcl-xL
1) Confidence 0.16 Published 2007 Journal BMC Cancer Section Body Doc Link PMC2222640 Disease Relevance 0.71 Pain Relevance 0
Bad is a pro-apoptotic member of the Bcl-2 family that can displace Bax from binding to Bcl-2 and Bcl-xL, resulting in cell death.
Bax Binding (binding) of Bcl-xL associated with apoptosis and death
2) Confidence 0.01 Published 2003 Journal Mol Cancer Section Body Doc Link PMC149414 Disease Relevance 1.32 Pain Relevance 0

General Comments

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