INT17296

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Context Info
Confidence 0.24
First Reported 1991
Last Reported 2002
Negated 1
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.29
Pain Relevance 0.65

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Acot1) mitochondrion (Acot1) lipid metabolic process (Acot1)
cytoplasm (Acot1)
Anatomy Link Frequency
substantia nigra 2
Acot1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Substantia nigra 1 100.00 Very High Very High Very High
Pain 4 99.52 Very High Very High Very High
antagonist 2 89.28 High High
Dopamine 3 88.84 High High
tetrodotoxin 1 85.60 High High
interneuron 1 49.76 Quite Low
dopamine receptor 1 33.92 Quite Low
Disease Link Frequency Relevance Heat
Pain 2 99.52 Very High Very High Very High
Nociception 1 94.96 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In males, exposure to the essential oil did not change the time course and magnitude of the ACh increase induced by pain.
Neg (not) Regulation (change) of Positive_regulation (increase) of ACh associated with pain
1) Confidence 0.24 Published 2002 Journal Neurosci. Lett. Section Abstract Doc Link 12213626 Disease Relevance 0.29 Pain Relevance 0.35
The second set of experiments tested the effects of 6-hydroxydopamine (6-OHDA) lesions of the substantia nigra on NT-induced increases of potassium-evoked ACh release.
Regulation (effects) of Positive_regulation (increases) of ACh in substantia nigra associated with substantia nigra
2) Confidence 0.13 Published 1991 Journal J. Neurochem. Section Abstract Doc Link 1899109 Disease Relevance 0 Pain Relevance 0.30

General Comments

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