INT173170

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Context Info
Confidence 0.06
First Reported 2003
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 7
Disease Relevance 3.11
Pain Relevance 0.47

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (PPA1) cytoplasm (PPA1)
Anatomy Link Frequency
T cells 3
hepatocytes 1
PPA1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 80 99.52 Very High Very High Very High
Inflammation 124 99.12 Very High Very High Very High
agonist 51 93.76 High High
Inflammatory mediators 22 60.76 Quite High
Bile 12 54.84 Quite High
chemokine 54 50.32 Quite High
cINOD 16 5.00 Very Low Very Low Very Low
rheumatoid arthritis 8 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
aspirin 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 197 100.00 Very High Very High Very High
INFLAMMATION 156 99.12 Very High Very High Very High
Infection 90 97.08 Very High Very High Very High
Peritonitis 45 91.28 High High
Liver Cancer 20 90.56 High High
Stress 16 88.00 High High
Hypolipidemia 4 86.32 High High
Hepatocellular Cancer 32 82.80 Quite High
Obesity 24 80.88 Quite High
Cancer 104 80.20 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Because the rodent hepatocarcinogenesis following PP exposure is mediated by PPAR?
Gene_expression (exposure) of PP
1) Confidence 0.06 Published 2003 Journal Comp Hepatol Section Body Doc Link PMC151270 Disease Relevance 0.73 Pain Relevance 0.04
The PP-induced suppression of apoptosis can be reproduced in cultured rodent hepatocytes with high concentrations of TNF?
Gene_expression (reproduced) of PP in hepatocytes associated with apoptosis
2) Confidence 0.06 Published 2003 Journal Comp Hepatol Section Body Doc Link PMC151270 Disease Relevance 0.74 Pain Relevance 0.03
Following PP exposure, the down-regulation of LF expression would result in increased levels of TNF?
Gene_expression (exposure) of PP
3) Confidence 0.06 Published 2003 Journal Comp Hepatol Section Body Doc Link PMC151270 Disease Relevance 0.10 Pain Relevance 0.13
For example, PP-induced expression of growth regulatory genes precedes entry of the cell in S phase [67].
Gene_expression (expression) of PP
4) Confidence 0.06 Published 2003 Journal Comp Hepatol Section Body Doc Link PMC151270 Disease Relevance 0.44 Pain Relevance 0.07
T cells, and where low numbers of bacteria produce trace amounts of HMB-PP.
Gene_expression (produce) of HMB-PP in T cells
5) Confidence 0.01 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2637987 Disease Relevance 0.24 Pain Relevance 0.13
2 T cells correlates with their ability to produce HMB-PP [15],[16].
Gene_expression (produce) of HMB-PP in T cells
6) Confidence 0.01 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2637987 Disease Relevance 0.15 Pain Relevance 0.05
2 T cells selectively recognize microbial pathogens capable of synthesizing HMB-PP (Table S3) [8]–[11], [14]–[16].
Gene_expression (synthesizing) of HMB-PP in T cells
7) Confidence 0.01 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2637987 Disease Relevance 0.72 Pain Relevance 0.03

General Comments

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