INT173399

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Context Info
Confidence 0.00
First Reported 2003
Last Reported 2003
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.18
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Ns5atp9) Golgi apparatus (GPER) endoplasmic reticulum (GPER)
nucleus (Ns5atp9) plasma membrane (GPER) signal transducer activity (GPER)
Anatomy Link Frequency
neutrophils 1
GPER (Homo sapiens)
Ns5atp9 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Inflammation 17 85.96 High High
halothane 4 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
Catecholamine 2 5.00 Very Low Very Low Very Low
corticosteroid 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
ischemia 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Adhesions 21 96.48 Very High Very High Very High
INFLAMMATION 19 85.96 High High
Asthma 3 5.00 Very Low Very Low Very Low
Hypersensitivity 3 5.00 Very Low Very Low Very Low
Necrosis 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Salivary Gland Disease 2 5.00 Very Low Very Low Very Low
Cancer 2 5.00 Very Low Very Low Very Low
Anaphylaxis 2 5.00 Very Low Very Low Very Low
Infection 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the rat feG (10-11M) reduced the binding of CD11b and CD16 antibodies to PAF-stimulated circulating neutrophils by 35% and 43%, respectively, and at 100 micrograms/kilograms intraperitoneally feG reduced neutrophil in vivo migration by 40%.
feG Binding (binding) of PAF in neutrophils
1) Confidence 0.00 Published 2003 Journal BMC Immunol Section Abstract Doc Link PMC152650 Disease Relevance 0.18 Pain Relevance 0.04

General Comments

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