INT175084

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Context Info
Confidence 0.61
First Reported 2003
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 11
Total Number 11
Disease Relevance 3.58
Pain Relevance 1.81

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
muscle 3
Mup1 (Mus musculus)
Pain Link Frequency Relevance Heat
carpal tunnel syndrome 327 99.40 Very High Very High Very High
Pain 337 98.04 Very High Very High Very High
addiction 4 81.60 Quite High
Inflammation 25 50.00 Quite Low
Inflammatory response 3 23.80 Low Low
medulla 2 21.68 Low Low
fibrosis 8 7.76 Low Low
Action potential 45 5.00 Very Low Very Low Very Low
Sciatica 21 5.00 Very Low Very Low Very Low
tolerance 9 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Carpal Tunnel Syndrome 333 99.40 Very High Very High Very High
Pain 331 98.04 Very High Very High Very High
Neuromuscular Disease 9 96.44 Very High Very High Very High
Muscle Disease 15 95.24 Very High Very High Very High
Aging 10 93.56 High High
Injury 27 87.88 High High
Stress 6 82.08 Quite High
Hypothermia 14 76.16 Quite High
Neuropathic Pain 63 73.76 Quite High
Motor Neuron Diseases 33 73.44 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the present study, the sEMG signal was described with four parameters derived from the IPL method: (1) the mean muscle CV which was the average of the obtained MUP velocities; the two statistical distribution variables, which were: (2) the within-subject MUP velocities’ skewness [Sk-peak velocity (PV)] and (3) the within-subject MUP velocities’ standard deviation (SD-PV), and (4) the peak frequency (PF), a variable expressing the amount of MUP activity (number of peaks = MUPs) per second.
Gene_expression (expressing) of MUP in muscle
1) Confidence 0.61 Published 2007 Journal Eur J Appl Physiol Section Body Doc Link PMC2039775 Disease Relevance 0.05 Pain Relevance 0.04
The long MUP durations of the severe CTS group relative to the mild and control groups again suggests that the severe group was undergoing or had undergone collateral reinnervation [5].
Gene_expression (durations) of MUP associated with carpal tunnel syndrome
2) Confidence 0.56 Published 2010 Journal J Neuroeng Rehabil Section Body Doc Link PMC2928769 Disease Relevance 0.73 Pain Relevance 0.57
To be included in the data set and therefore in subsequent analyses, a SMUP had to be temporally aligned (within 10 ms) with its corresponding MUP and verified as a distinct waveform with respect to the RMS of the signal baseline.


Gene_expression (aligned) of MUP
3) Confidence 0.56 Published 2010 Journal J Neuroeng Rehabil Section Body Doc Link PMC2928769 Disease Relevance 0 Pain Relevance 0
As such, the larger MUP amplitude in the severe CTS group as compared to the mild CTS and control groups suggests that larger motor units were active during EMG signal detection which in turn may suggest that collateral sprouting may have occurred at some point prior to the study.
Gene_expression (amplitude) of MUP associated with carpal tunnel syndrome
4) Confidence 0.56 Published 2010 Journal J Neuroeng Rehabil Section Body Doc Link PMC2928769 Disease Relevance 0.52 Pain Relevance 0.37
In the present study, the sEMG signal was described with four parameters derived from the IPL method: (1) the mean muscle CV which was the average of the obtained MUP velocities; the two statistical distribution variables, which were: (2) the within-subject MUP velocities’ skewness [Sk-peak velocity (PV)] and (3) the within-subject MUP velocities’ standard deviation (SD-PV), and (4) the peak frequency (PF), a variable expressing the amount of MUP activity (number of peaks = MUPs) per second.
Gene_expression (expressing) of MUP in muscle
5) Confidence 0.46 Published 2007 Journal Eur J Appl Physiol Section Body Doc Link PMC2039775 Disease Relevance 0.06 Pain Relevance 0.04
The MUP frequency variable, expressing the amount of MU activity produced as a result of recruitment and rate coding did not change during these tests.
Gene_expression (expressing) of MUP
6) Confidence 0.42 Published 2007 Journal Eur J Appl Physiol Section Body Doc Link PMC2039775 Disease Relevance 0.13 Pain Relevance 0
The shorter MUP duration and smaller AAR values found in the control group are not consistent with our speculation that NSAP may be caused by a myopathic process.
Gene_expression (duration) of MUP associated with pain
7) Confidence 0.34 Published 2008 Journal J Neuroeng Rehabil Section Body Doc Link PMC2654455 Disease Relevance 0.85 Pain Relevance 0.26
Decreased MUP duration and reduced AAR values are important distinguishing features of needle-detected MUPs in neuromuscular disease, and in myopathic disease the MUPs are expected to be shorter in duration and have smaller AAR values than in unaffected muscles [48].
Gene_expression (duration) of MUP in muscles associated with neuromuscular disease and muscle disease
8) Confidence 0.34 Published 2008 Journal J Neuroeng Rehabil Section Body Doc Link PMC2654455 Disease Relevance 0.75 Pain Relevance 0.25
-barrel structure of LCN6 closely matches that of Mup1, however the relatively short C-terminus of human LCN6 lacks the region that in Mup1 contains the cysteine that forms a disulfide bond with a cysteine in the ?
Gene_expression (matches) of Mup1
9) Confidence 0.32 Published 2003 Journal Reprod Biol Endocrinol Section Body Doc Link PMC293424 Disease Relevance 0 Pain Relevance 0
Independent MUP analysis
Gene_expression (analysis) of MUP
10) Confidence 0.30 Published 2010 Journal J Neuroeng Rehabil Section Body Doc Link PMC2848053 Disease Relevance 0.33 Pain Relevance 0.27
In addition to genes involved in complement and coagulation pathways, and cytochrome p450 genes, there are several genes of the mouse major urinary protein family, or Mup, found in this down-regulated cluster.
Gene_expression (found) of Mup
11) Confidence 0.22 Published 2010 Journal BMC Neurosci Section Body Doc Link PMC2939656 Disease Relevance 0.16 Pain Relevance 0

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