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Context Info
Confidence 0.00
First Reported 2003
Last Reported 2003
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.20
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endoplasmic reticulum (RASGRP1, MAP3K5) cytoplasm (RASGRP1, MAP3K5) cell differentiation (RASGRP1)
Golgi apparatus (RASGRP1) response to stress (MAP3K5) cytosol (RASGRP1)
Anatomy Link Frequency
epithelial cells 1
RASGRP1 (Homo sapiens)
MAP3K5 (Homo sapiens)
Pain Link Frequency Relevance Heat
Somatostatin 2 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Adenocarcinoma 10 97.44 Very High Very High Very High
Hyperplasia 2 94.48 High High
Cancer 86 90.96 High High
Genomic Instability 9 90.12 High High
Lung Cancer 76 84.08 Quite High
Targeted Disruption 3 82.16 Quite High
Squamous Cell Carcinoma 9 57.12 Quite High
Carcinoma 2 54.40 Quite High
Congenital Anomalies 20 50.00 Quite Low
Nicotine Addiction 44 38.88 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
K-ras, once mutated (most frequently codon 12 G-T transversions), can transform airway epithelial cells [18,19] by activating the ERK-MAP kinase pathway.
ras Positive_regulation (activating) of ERK-MAP kinase in epithelial cells
1) Confidence 0.00 Published 2003 Journal Respir Res Section Body Doc Link PMC314397 Disease Relevance 1.20 Pain Relevance 0

General Comments

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