INT177651

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Context Info
Confidence 0.04
First Reported 2004
Last Reported 2004
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.43
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Vmn1r51) plasma membrane (Vmn1r51) signal transducer activity (Vmn1r51)
Vmn1r51 (Mus musculus)
Pain Link Frequency Relevance Heat
Catecholamine 7 52.76 Quite High
anesthesia 4 5.00 Very Low Very Low Very Low
antagonist 3 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
noradrenaline 1 5.00 Very Low Very Low Very Low
Neurotransmitter 1 5.00 Very Low Very Low Very Low
Kinase C 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 29 98.74 Very High Very High Very High
Shock 34 56.88 Quite High
Sepsis 13 49.40 Quite Low
Pressure Volume 2 Under Development 4 37.28 Quite Low
Hemorrhagic Shock 2 24.96 Low Low
Hypoxia 5 21.44 Low Low
Vasoplegia 1 7.08 Low Low
Myocardial Infarction 12 5.00 Very Low Very Low Very Low
Heart Rate Under Development 5 5.00 Very Low Very Low Very Low
Stroke 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Whether vasopressin causes vasoconstriction or vasodilation depends on the vascular bed studied [46], which may, in turn, depend on the receptor density (V1R versus OTR), the model studied, the dose of vasopressin [47], and the duration of exposure to the hormone [48].
Spec (may) Regulation (depend) of V1R associated with increased venous pressure under development
1) Confidence 0.04 Published 2004 Journal Crit Care Section Body Doc Link PMC420051 Disease Relevance 0.43 Pain Relevance 0.03

General Comments

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