INT177934

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Context Info
Confidence 0.19
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.23
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

pigmentation (NF1) signal transduction (NF1) nucleus (NF1)
intracellular (NF1) extracellular matrix organization (NF1) cytoplasm (NF1)
NF1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 6 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Watson Syndrome 19 100.00 Very High Very High Very High
Cancer 16 90.16 High High
Nerve Sheath Neoplasms 22 78.56 Quite High
Reprotox - General 1 2 74.24 Quite High
Apoptosis 37 73.28 Quite High
Malignant Neoplastic Disease 6 5.00 Very Low Very Low Very Low
Familial Adenomatous Polyposis 5 5.00 Very Low Very Low Very Low
Death 3 5.00 Very Low Very Low Very Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Increased levels of RAS-GTP have previously been shown in MPNSTs from NF1 patients [29-31], caused perhaps by loss of the RAS-GAP-function of the NF1 gene product, neurofibromin.
Regulation (function) of Gene_expression (product) of NF1 associated with watson syndrome
1) Confidence 0.19 Published 2004 Journal Cancer Cell Int Section Body Doc Link PMC425591 Disease Relevance 1.23 Pain Relevance 0

General Comments

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