INT178798

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Context Info
Confidence 0.42
First Reported 2004
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 1.71
Pain Relevance 0.74

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Camp) extracellular region (Camp) cytoplasm (Camp)
Anatomy Link Frequency
peritoneal cavity 1
leukocyte 1
CLP 1
Camp (Mus musculus)
Pain Link Frequency Relevance Heat
lidocaine 102 97.16 Very High Very High Very High
cytokine 64 91.48 High High
anesthesia 3 81.92 Quite High
Inflammation 135 46.00 Quite Low
chemokine 23 25.28 Quite Low
Inflammatory response 47 7.12 Low Low
isoflurane 35 5.00 Very Low Very Low Very Low
local anesthetic 6 5.00 Very Low Very Low Very Low
agonist 6 5.00 Very Low Very Low Very Low
aspirin 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cramps 11 100.00 Very High Very High Very High
Body Weight 4 93.00 High High
Peritonitis 74 91.20 High High
Sepsis 87 88.16 High High
Lifespan 2 76.88 Quite High
Diarrhoea 1 61.36 Quite High
Urological Neuroanatomy 48 56.08 Quite High
INFLAMMATION 182 46.00 Quite Low
Endotoxemia 15 36.04 Quite Low
Neutropenia 6 20.44 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, exudate CRAMP/LL-37 may also contribute to increased PMN numbers obtained at 4 h in lidocaine-treated mice (Fig. 1A).
Gene_expression (exudate) of CRAMP associated with cramps and lidocaine
1) Confidence 0.42 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2268966 Disease Relevance 0.29 Pain Relevance 0.52
The animals were initially divided into 4 groups that were treated by subcutaneous route 6 hours before the CLP induction.
Gene_expression (induction) of CLP in CLP
2) Confidence 0.42 Published 2008 Journal BMC Complement Altern Med Section Body Doc Link PMC2571085 Disease Relevance 0.41 Pain Relevance 0.08
Similar to the CLP response, the leukocyte influx into the peritoneal cavities in response to glycogen consisted predominantly of neutrophils (Figures 2B, 3).
Gene_expression (response) of CLP in leukocyte
3) Confidence 0.36 Published 2004 Journal BMC Clin Pathol Section Body Doc Link PMC503395 Disease Relevance 0.15 Pain Relevance 0
Although a fewer number of neutrophils were present in the peritoneal cavities of P/I null mice, the ratio of neutrophil infiltration (i.e. ratio = infiltrated peritoneal neutrophils in response to CLP/neutrophils normally present in peritoneal cavity of the control mouse) was significantly higher in the P/I null mice than those of wild-type mice.
Gene_expression (neutrophils) of CLP in peritoneal cavity
4) Confidence 0.36 Published 2004 Journal BMC Clin Pathol Section Body Doc Link PMC503395 Disease Relevance 0.13 Pain Relevance 0
2, P<0.01) and KC (26.5±13.5% of positive controls, “CLP+Saline”; versus 11.4±8.6% of positive controls, “CLP+EGCG”; n?
Gene_expression (controls) of CLP
5) Confidence 0.08 Published 2007 Journal PLoS ONE Section Body Doc Link PMC2048740 Disease Relevance 0.74 Pain Relevance 0.13

General Comments

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