INT179232

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (CREM) mitochondrion (GSR) small molecule metabolic process (GSR)
DNA binding (CREM) cytoplasm (GSR) cytosol (GSR)
CREM (Homo sapiens)
GSR (Homo sapiens)
Pain Link Frequency Relevance Heat
chemokine 1 61.36 Quite High
cytokine 11 60.40 Quite High
Inflammation 17 38.88 Quite Low
agonist 3 31.68 Quite Low
cva 5 5.00 Very Low Very Low Very Low
opiate 3 5.00 Very Low Very Low Very Low
medulla 3 5.00 Very Low Very Low Very Low
dexamethasone 3 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
Catecholamine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Sepsis 33 50.00 Quite Low
INFLAMMATION 19 38.88 Quite Low
Apoptosis 2 36.16 Quite Low
Repression 1 33.36 Quite Low
Cv General 2 Under Development 5 6.84 Low Low
Necrosis 5 5.00 Very Low Very Low Very Low
Adrenal Insufficiency 5 5.00 Very Low Very Low Very Low
Cv General 3 Under Development 5 5.00 Very Low Very Low Very Low
Secondary Adrenal Insufficiency 4 5.00 Very Low Very Low Very Low
Sleep Disorders 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The GR-induced transrepression occurs through DNA-dependent mechanisms (i.e. displacement of an activator, overlapping binding sites, or binding to continuous negative glucocorticoid responsive element) and via DNA independent mechanisms (without direct contact between the GR and DNA).
glucocorticoid responsive element Binding (binding) of GR-induced
1) Confidence 0.00 Published 2004 Journal Crit Care Section Body Doc Link PMC522845 Disease Relevance 0 Pain Relevance 0.06

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox