INT179754

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Context Info
Confidence 0.34
First Reported 2004
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.93
Pain Relevance 1.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (SLC1A2) transmembrane transport (SLC1A2)
Anatomy Link Frequency
plasma 2
SLC1A2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Kinase C 12 98.96 Very High Very High Very High
Glutamate 190 92.80 High High
cytokine 40 88.92 High High
adenocard 4 37.12 Quite Low
Central nervous system 25 25.92 Quite Low
addiction 1 8.40 Low Low
Multiple sclerosis 22 5.00 Very Low Very Low Very Low
Spinal cord 15 5.00 Very Low Very Low Very Low
cerebral cortex 14 5.00 Very Low Very Low Very Low
depression 12 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neuromyelitis Optica 87 100.00 Very High Very High Very High
Targeted Disruption 5 76.84 Quite High
Alzheimer's Dementia 2 47.52 Quite Low
Fatigue 96 12.24 Low Low
Cancer 6 10.56 Low Low
Necrosis 7 10.08 Low Low
Stress 10 8.96 Low Low
Cognitive Disorder 14 6.04 Low Low
Demyelinating Disease 29 5.00 Very Low Very Low Very Low
Disease 20 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We excluded the possibility that NMO serum might contain EAAT2-specific IgG in addition to AQP4 IgG, by testing the effect of NMO serum on plasma membrane expression of EAAT2 in HEK-293 cells transfected with AQP5-GFP (Fig. 4 C).
Regulation (effect) of Gene_expression (expression) of EAAT2 in plasma associated with neuromyelitis optica
1) Confidence 0.34 Published 2008 Journal The Journal of Experimental Medicine Section Body Doc Link PMC2571922 Disease Relevance 0.77 Pain Relevance 0.06
The synthesis of GLT-1 has been shown to be stimulated by factors acting via receptor tyrosine kinases and pathways dependent on phosphatidylinositol-3-kinase (PI3K) and the nuclear transcription factor NF?
Regulation (stimulated) of Gene_expression (synthesis) of GLT-1
2) Confidence 0.08 Published 2004 Journal J Neuroinflammation Section Body Doc Link PMC533886 Disease Relevance 0.08 Pain Relevance 0.61
The synthesis of GLT-1 has been shown to be stimulated by factors acting via receptor tyrosine kinases and pathways dependent on phosphatidylinositol-3-kinase (PI3K) and the nuclear transcription factor NF?
Regulation (pathways) of Gene_expression (synthesis) of GLT-1
3) Confidence 0.08 Published 2004 Journal J Neuroinflammation Section Body Doc Link PMC533886 Disease Relevance 0.08 Pain Relevance 0.65

General Comments

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