INT180536

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Context Info
Confidence 0.45
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.13
Pain Relevance 0.28

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
brain 2
CR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Glutamate 1 99.28 Very High Very High Very High
ischemia 36 85.60 High High
Inflammation 7 78.20 Quite High
chemokine 1 51.72 Quite High
cva 4 51.20 Quite High
COX-2 inhibitor 12 38.32 Quite Low
anesthesia 3 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 1 99.66 Very High Very High Very High
Targeted Disruption 1 99.40 Very High Very High Very High
Brain Injury 3 98.90 Very High Very High Very High
Cv General 4 Under Development 9 86.32 High High
Injury 5 83.36 Quite High
INFLAMMATION 7 78.20 Quite High
Stroke 37 75.84 Quite High
Cv Unclassified Under Development 20 74.00 Quite High
Adhesions 1 58.32 Quite High
Cv General 3 Under Development 4 51.20 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Several recent studies have demonstrated a marked neuroprotective effect of nimesulide on chronic cerebral hypoperfusion [15], kainate-induced excitotoxicity [16], quisqualic acid-induced neurodegeneration [17], diffuse traumatic brain injury [18,19], glutamate-mediated apoptotic damage [20] and induction of the expression of the B subunit of endogenous complement component C1q (C1qB) in transgenic mice with neuronal overexpression of human COX-2 [21].
Positive_regulation (induction) of Gene_expression (expression) of complement component in brain associated with targeted disruption, glutamate, apoptosis and brain injury
1) Confidence 0.45 Published 2005 Journal J Neuroinflammation Section Body Doc Link PMC546225 Disease Relevance 1.13 Pain Relevance 0.28

General Comments

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