INT180809

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Context Info
Confidence 0.03
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.84
Pain Relevance 1.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Sorbs1) aging (Ngf) cytoplasm (Sorbs1)
cytosol (Sorbs1) extracellular space (Ngf) signal transduction (Ngf)
Ngf (Rattus norvegicus)
Sorbs1 (Mus musculus)
Pain Link Frequency Relevance Heat
Nerve growth factor 100 100.00 Very High Very High Very High
qutenza 52 100.00 Very High Very High Very High
Eae 1 95.00 High High
Hyperalgesia 3 93.48 High High
Thermal hyperalgesia 3 93.24 High High
Calcitonin gene-related peptide 96 89.40 High High
substance P 5 89.00 High High
Neuropeptide 11 87.40 High High
Inflammation 6 86.88 High High
dorsal root ganglion 12 63.64 Quite High
Disease Link Frequency Relevance Heat
Injury 23 95.00 High High
Hyperalgesia 7 93.48 High High
INFLAMMATION 5 86.88 High High
Nociception 8 83.32 Quite High
Ganglion Cysts 73 63.64 Quite High
Hypersensitivity 2 31.44 Quite Low
Pain 3 20.76 Low Low
Headache 6 5.00 Very Low Very Low Very Low
Apoptosis 2 5.00 Very Low Very Low Very Low
Nervous System Injury 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In accordance with this increased CAP sensitivity, NGF increases the expression of the CAP- and noxious heat-sensitive ion channel vanilloid receptor type 1 (TRPV1) through the ras/p38 MAP kinase signaling pathway, thereby promoting thermal hyperalgesia [25,26].
NGF Positive_regulation (increases) of Positive_regulation (increased) of CAP associated with nerve growth factor, qutenza and thermal hyperalgesia
1) Confidence 0.03 Published 2005 Journal BMC Neurosci Section Body Doc Link PMC548274 Disease Relevance 0.84 Pain Relevance 1.40

General Comments

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