INT182091

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Context Info
Confidence 0.02
First Reported 2004
Last Reported 2004
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.61
Pain Relevance 0.78

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

MRXS5 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 10 99.62 Very High Very High Very High
Analgesic 2 96.48 Very High Very High Very High
Inflammation 9 93.96 High High
Inflammatory response 3 81.40 Quite High
cytokine 25 67.12 Quite High
Pain 2 59.88 Quite High
Inflammatory mediators 2 34.16 Quite Low
COX-2 inhibitor 6 6.48 Low Low
diclofenac 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 15 93.96 High High
Infection 5 75.36 Quite High
Pain 2 59.88 Quite High
Fever 6 59.52 Quite High
Sepsis 51 50.52 Quite High
Systemic Inflammatory Response Syndrome 2 31.16 Quite Low
Death 1 25.08 Quite Low
Cancer 4 5.00 Very Low Very Low Very Low
Necrosis 3 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The primary pharmacological action of NSAIDs is, of course, to decrease the formation of PGs and thromboxanes by inhibiting COX, a key enzyme in the biochemical pathway that leads to formation of these potent mediators [24].
Negative_regulation (decrease) of PGs Binding (formation) of associated with cinod
1) Confidence 0.02 Published 2004 Journal Crit Care Section Body Doc Link PMC1065065 Disease Relevance 0.61 Pain Relevance 0.78

General Comments

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