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Context Info
Confidence 0.32
First Reported 2004
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 2.40
Pain Relevance 0.29

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (AGTR2) signal transducer activity (AGTR2)
AGTR2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 88 83.60 Quite High
Inflammatory mediators 5 60.24 Quite High
cytokine 11 57.68 Quite High
chemokine 5 56.80 Quite High
fibrosis 6 53.28 Quite High
agonist 19 50.00 Quite Low
cINOD 3 49.12 Quite Low
adenocard 1 27.00 Quite Low
antagonist 7 20.92 Low Low
ischemia 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetic Retinopathy 47 96.44 Very High Very High Very High
Diabetes Mellitus 30 92.08 High High
Vasculitis 2 91.08 High High
INFLAMMATION 93 83.60 Quite High
Cancer 91 80.40 Quite High
Atherosclerosis 3 76.48 Quite High
Disease 35 74.96 Quite High
Increased Venous Pressure Under Development 9 73.68 Quite High
Injury 10 73.36 Quite High
Metastasis 7 70.36 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
For instance, the associations of ACE and AGTR2 with LV mass were reported to vary according to dietary salt intake in one investigation [48].
AGTR2 Binding (associations) of
1) Confidence 0.32 Published 2007 Journal BMC Med Genet Section Body Doc Link PMC1995617 Disease Relevance 0 Pain Relevance 0
In this work [65], the AT1 and AT2 receptors are recognized as having a substantial role in the tissue repair and healing processes of injured arteries.
AT2 Binding (recognized) of
2) Confidence 0.19 Published 2004 Journal J Inflamm (Lond) Section Body Doc Link PMC1074345 Disease Relevance 1.23 Pain Relevance 0.20
Furthermore, AT1 and AT2, AII, and its
AT2 Binding (its) of
3) Confidence 0.08 Published 2008 Journal PPAR Research Section Body Doc Link PMC2396446 Disease Relevance 1.16 Pain Relevance 0.09

General Comments

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