INT183425

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Context Info
Confidence 0.09
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.84
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Pde2a) signal transduction (Pde2a) Golgi apparatus (Pde2a)
endoplasmic reticulum (Pde2a) plasma membrane (Pde2a) nucleus (Pde2a)
Pde2a (Rattus norvegicus)
Pain Link Frequency Relevance Heat
fibrosis 2 92.64 High High
ischemia 1 14.28 Low Low
adenocard 4 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 9 99.92 Very High Very High Very High
Coronary Heart Disease 20 95.48 Very High Very High Very High
Hypertrophy 5 93.28 High High
Cardiovascular Disorder Under Development 1 92.64 High High
Pressure And Volume Under Development 2 91.92 High High
Natriuresis 4 76.12 Quite High
Reprotox - General 2 3 76.00 Quite High
Cv Unclassified Under Development 1 14.28 Low Low
Fibrosis 1 5.00 Very Low Very Low Very Low
Heart Rate Under Development 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Inhibition of cGMP degradation by sildenafil with a consequent accumulation of this signaling molecule may act as a negative regulator against cardiac hypertrophy in-vivo.
Negative_regulation (Inhibition) of Protein_catabolism (degradation) of cGMP associated with coronary heart disease
1) Confidence 0.09 Published 2005 Journal BMC Pharmacol Section Body Doc Link PMC1131906 Disease Relevance 0.84 Pain Relevance 0.05

General Comments

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