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Context Info
Confidence 0.33
First Reported 2005
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.01
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (BCL2) cell morphogenesis (BCL2) endoplasmic reticulum (BCL2)
intracellular (BCL2) cytoplasm (BCL2) cytosol (BCL2)
BCL2 (Homo sapiens)
Pain Link Frequency Relevance Heat
lidocaine 1 5.00 Very Low Very Low Very Low
methotrexate 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
local anesthetic 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 7 99.20 Very High Very High Very High
Apoptosis 115 98.32 Very High Very High Very High
Colon Cancer 43 77.04 Quite High
Carcinoma 1 70.52 Quite High
Reprotox - General 1 10 63.04 Quite High
Breast Cancer 7 54.64 Quite High
Cancer 24 47.28 Quite Low
Hyperplasia 4 32.88 Quite Low
Chronic Lymphoid Leukemia 1 18.72 Low Low
Thyroid Neoplasm 1 14.32 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Inhibition of miR-34a in Par-4-overexpressing cells resulted in an up-regulation of BCL2 protein with a corresponding decrease in apoptotic sensitivity to 5-FU.
Negative_regulation (Inhibition) of Regulation (regulation) of BCL2 associated with apoptosis
1) Confidence 0.33 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2883962 Disease Relevance 0.47 Pain Relevance 0
Furthermore, as discussed by Ferrieres and colleagues [50], these results suggest a loss in the control of Bcl-2 by progesterone in diseases originating from epithelial lobular components.
Negative_regulation (loss) of Regulation (control) of Bcl-2 associated with disease
2) Confidence 0.29 Published 2005 Journal Breast Cancer Res Section Body Doc Link PMC1143573 Disease Relevance 0.53 Pain Relevance 0

General Comments

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