INT183790

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Context Info
Confidence 0.04
First Reported 2005
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.97
Pain Relevance 1.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (TBK1) plasma membrane (TBK1) cytoplasm (TBK1)
Anatomy Link Frequency
mononuclear cells 1
TBK1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 50 99.76 Very High Very High Very High
Inflammation 176 99.36 Very High Very High Very High
metalloproteinase 3 87.04 High High
cINOD 37 77.96 Quite High
tolerance 2 74.80 Quite High
Pain 22 72.60 Quite High
cva 3 61.44 Quite High
aspirin 9 56.40 Quite High
antagonist 2 45.16 Quite Low
withdrawal 1 27.48 Quite Low
Disease Link Frequency Relevance Heat
INFLAMMATION 213 99.36 Very High Very High Very High
Injury 15 97.52 Very High Very High Very High
Diabetes Mellitus 17 90.44 High High
Obesity 17 89.64 High High
Aging 29 89.04 High High
Stress 5 86.72 High High
Overnutrition 3 83.88 Quite High
Arthropathy 2 79.44 Quite High
Impaired Glucose Tolerance 2 74.80 Quite High
Insulin Resistance 9 72.88 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The NF-kB proteins are localized in the cytoplasm of the cell and are associated with a family of inhibitory proteins known as inhibitor of kB (IkB).[43119] The TNF-?
NF-kB Binding (associated) of
1) Confidence 0.04 Published 2010 Journal Surgical Neurology International Section Body Doc Link PMC3011108 Disease Relevance 1.22 Pain Relevance 0.84
In fact, insulin reduces ROS generation by mononuclear cells, suppresses NADPH oxidase expression and intranuclear NF-kB binding, induces IkB expression and suppresses some inflammatory molecules [69,76].
NF-kB Binding (binding) of in mononuclear cells associated with inflammation
2) Confidence 0.03 Published 2005 Journal Immun Ageing Section Body Doc Link PMC1166571 Disease Relevance 1.75 Pain Relevance 0.32

General Comments

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