INT185142

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Context Info
Confidence 0.01
First Reported 2005
Last Reported 2005
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.22

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Rnpep) cytoplasm (Snrpd1) extracellular space (Rnpep)
peptidase activity (Rnpep) extracellular region (Rnpep) mRNA processing (Snrpd1)
Rnpep (Mus musculus)
Snrpd1 (Mus musculus)
Pain Link Frequency Relevance Heat
Action potential 1 72.12 Quite High
Kinase C 57 68.32 Quite High
antagonist 4 62.24 Quite High
conotoxin 3 60.52 Quite High
Calcium channel 3 22.28 Low Low
Clonidine 26 5.00 Very Low Very Low Very Low
tetrodotoxin 3 5.00 Very Low Very Low Very Low
Neurotransmitter 3 5.00 Very Low Very Low Very Low
agonist 3 5.00 Very Low Very Low Very Low
potassium channel 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 10 46.28 Quite Low
Overdose 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The EFS-induced SMD remained unaffected by the InsP3 receptor inhibitors 2APB and xestospongin C, ruling out a substantial role of InsP3 receptor-mediated Ca2+ release in this response.
APB Neg (unaffected) Regulation (unaffected) of SMD
1) Confidence 0.01 Published 2005 Journal BMC Physiol Section Body Doc Link PMC1183225 Disease Relevance 0 Pain Relevance 0.22

General Comments

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