INT185222

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Context Info
Confidence 0.60
First Reported 2005
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 7
Disease Relevance 4.98
Pain Relevance 0.64

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (EBAG9) plasma membrane (EBAG9)
Anatomy Link Frequency
immune system 4
immune cells 2
bone marrow 2
EBAG9 (Homo sapiens)
Pain Link Frequency Relevance Heat
endometriosis 156 99.76 Very High Very High Very High
tolerance 25 96.36 Very High Very High Very High
Dismenorea 10 5.00 Very Low Very Low Very Low
Immobilon 4 5.00 Very Low Very Low Very Low
pain pelvic 3 5.00 Very Low Very Low Very Low
Pain 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Endometriosis 270 99.76 Very High Very High Very High
Endometrial Cancer 3 98.28 Very High Very High Very High
Cicatrix 141 96.84 Very High Very High Very High
Abruptio Placentae 8 96.80 Very High Very High Very High
Cancer 31 95.68 Very High Very High Very High
Death 14 92.96 High High
Apoptosis 37 92.52 High High
Fetal Death 28 91.16 High High
Ovarian Cysts 3 87.00 High High
Reprotox - General 3 50 86.92 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The alterations of RCAS1 protein expression in placental cells seem to determine the beginning of the labor and participate in the placental abruption.
Regulation (alterations) of Gene_expression (expression) of RCAS1 associated with abruptio placentae
1) Confidence 0.60 Published 2005 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC1184098 Disease Relevance 0.27 Pain Relevance 0
Our results indicate that alterations in RCAS1 expression may be involved in the changes of maternal immune system that take place during stillbirth.


Regulation (alterations) of Gene_expression (expression) of RCAS1 in immune system
2) Confidence 0.60 Published 2005 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1184098 Disease Relevance 0.41 Pain Relevance 0.05
The expression of RCAS1 has been detected in the bone marrow, the Waldeyer's ring, the placenta, the endometrium, and the tubal mucosa thus indicating its role in immune cells regulation [11-13].
Regulation (detected) of Gene_expression (expression) of RCAS1 in bone marrow
3) Confidence 0.41 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1574328 Disease Relevance 0.83 Pain Relevance 0.06
In our previous report a significantly lower RCAS1 expression was noticed in ovarian endometriosis in comparison to endometrial carcinoma [10].
Regulation (noticed) of Gene_expression (expression) of RCAS1 associated with endometriosis and endometrial cancer
4) Confidence 0.41 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1574328 Disease Relevance 1.16 Pain Relevance 0.17
Conclusions: These preliminary results indicate that the alterations of RCAS1 expression in the human placenta may be involved in the changes of maternal immune system that take place during stillbirth.



Regulation (alterations) of Gene_expression (expression) of RCAS1 in immune system
5) Confidence 0.27 Published 2005 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC1184098 Disease Relevance 0.27 Pain Relevance 0
The alterations of RCAS1 protein expression in placental cells seem to determine the onset of labor and participate in the placental abruption [5,6].
Regulation (alterations) of Gene_expression (expression) of RCAS1 associated with abruptio placentae
6) Confidence 0.27 Published 2005 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1184098 Disease Relevance 0.64 Pain Relevance 0.03
The alterations in RCAS1 and Metallothionein expression are accompanied by changes in CD25, CD56 and CD69 antigens expression on immune cells.
Regulation (alterations) of Gene_expression (expression) of RCAS1 in immune cells
7) Confidence 0.25 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1574328 Disease Relevance 1.39 Pain Relevance 0.34

General Comments

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