INT185793

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Context Info
Confidence 0.16
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.33
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (SPP1, COL1A2) extracellular region (SPP1, COL1A2) cell adhesion (SPP1)
protein binding, bridging (COL1A2) cytoplasm (SPP1)
Anatomy Link Frequency
epithelial cells 1
SPP1 (Homo sapiens)
COL1A2 (Homo sapiens)
Pain Link Frequency Relevance Heat
fibrosis 72 74.04 Quite High
cytokine 5 65.00 Quite High
Inflammation 3 5.00 Very Low Very Low Very Low
corticosteroid 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pulmonary Fibrosis 65 74.04 Quite High
Adhesions 3 67.72 Quite High
Fibrosis 9 43.12 Quite Low
Internal Fibrosis 1 37.44 Quite Low
Keloid Scars 1 29.36 Quite Low
Myocardial Infarction 1 20.40 Low Low
Disease 6 5.00 Very Low Very Low Very Low
Pulmonary Disease 3 5.00 Very Low Very Low Very Low
Hyperplasia 2 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, in fibroblasts, osteopontin increased TIMP-1 and type I collagen and inhibited MMP-1 expression, while in alveolar epithelial cells it induced MMP-7.
osteopontin Positive_regulation (increased) of type I collagen in epithelial cells
1) Confidence 0.16 Published 2005 Journal PLoS Medicine Section Body Doc Link PMC1198037 Disease Relevance 0.33 Pain Relevance 0.16

General Comments

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