INT185825
From wiki-pain
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Sentences Mentioned In
Key: | Protein | Mutation | Event | Anatomy | Negation | Speculation | Pain term | Disease term |
Although the level of MMP7 secretion was the highest in the secretory phase, the difference from the proliferative phase did not reach statistical significance, neither in the endometrium nor in the fallopian tube. | |||||||||||||||
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Matrix metalloproteinase 7 (MMP7) is secreted mostly from the endometrial epithelium cells where attachment occurs and is suggested to be important in the implantation process [13]. | |||||||||||||||
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Although the level of MMP7 secretion was the highest in the secretory phase the difference from the proliferative phase did not reach statistical significance, neither in the endometrium nor in the fallopian tube. | |||||||||||||||
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Matrix metalloproteinase 7 (MMP7) is secreted mostly from the endometrial epithelium cells where attachment occurs and is suggested to be important in the implantation process [13]. | |||||||||||||||
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This is also supported by the colocalization of MMP-7 and osteopontin in IPF epithelial cells, and by the computational relationship of the expression levels of osteopontin and MMP-7. | |||||||||||||||
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Interestingly, osteopontin was colocalized with MMP-7 in alveolar epithelial cells of IPF lungs, and application of the weakest link models to microarray data suggested that the genes of both interacted to affect the IPF phenotype. | |||||||||||||||
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-catenin nuclear localization in proliferative bronchiolar lesions, where it colocalized with MMP-7 [34]. | |||||||||||||||
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Although eEPCs were grouped with monocytes, some subtle mRNA transcription differences were identified such as complement components (C1QC, C1QB), matrix metalloproteinases (MMP9, MMP7, ADAMDEC1) and osteopontin (SPP1) which were higher in eEPCs than in monocytes. | |||||||||||||||
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Interestingly, expression levels of genes encoding secreted proteins from different functional groups including CFI, CC2, NRG1, MMP7, WNT5A, RSOPO2, CSF3R, DPP4, IL1 were deregulated altered in CHM cells compared to the control. | |||||||||||||||
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These redox based natural agents appear to limit cartilage breakdown by blocking the expression, production and release of matrix metalloproteases by activated chondrocytes via a NF-? | |||||||||||||||
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General Comments
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