INT18594

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Context Info
Confidence 0.11
First Reported 1990
Last Reported 1990
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 1.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Acot1) mitochondrion (Acot1) oxidoreductase activity (Fdft1)
endoplasmic reticulum (Fdft1) transferase activity, transferring alkyl or aryl (other than methyl) groups (Fdft1) lipid metabolic process (Acot1)
Fdft1 (Rattus norvegicus)
Acot1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Somatostatin 22 100.00 Very High Very High Very High
antagonist 2 98.28 Very High Very High Very High
Hippocampus 4 92.44 High High
Calcium channel 2 84.64 Quite High
conotoxin 1 81.92 Quite High
tetrodotoxin 1 70.24 Quite High
Neuropeptide 1 43.76 Quite Low

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The increased ACh release in the presence of either peptide appeared to be mediated by an interaction with SS receptors because cyclo-SS, a putative SS antagonist, abolished the effects of both SS-28 and SS-14.
SS Spec (625) Binding (interaction) of Positive_regulation (mediated) of Localization (release) of ACh associated with antagonist and somatostatin
1) Confidence 0.11 Published 1990 Journal J. Neurochem. Section Abstract Doc Link 1976754 Disease Relevance 0 Pain Relevance 1.11

General Comments

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