INT18637

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Context Info
Confidence 0.39
First Reported 1990
Last Reported 2007
Negated 1
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 0.15
Pain Relevance 0.61

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

OAP (Homo sapiens)
Pain Link Frequency Relevance Heat
Clonidine 2 99.44 Very High Very High Very High
antagonist 4 92.48 High High
cytokine 40 84.92 Quite High
dexamethasone 7 81.12 Quite High
Inflammation 34 74.20 Quite High
agonist 14 61.92 Quite High
Crohn's disease 7 5.00 Very Low Very Low Very Low
corticosteroid 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Inflammatory Bowel Disease 32 80.08 Quite High
INFLAMMATION 35 74.20 Quite High
Disease 8 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Recurrence 2 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low
Necrosis 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Colitis 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While clonidine (0.5 to 10 microM) did not affect OAP or induction of triggered activity, yohimbine (2 microM) decreased the amplitude of OAP and abolished triggered activity induced by either acetylstrophanthidin or high Ca2+.
Neg (not) Regulation (affect) of OAP associated with clonidine
1) Confidence 0.39 Published 1990 Journal J. Mol. Cell. Cardiol. Section Abstract Doc Link 1977923 Disease Relevance 0 Pain Relevance 0.20
Thus, specific alpha 1-adrenergic actions of phenylephrine may exert either pro-arrhythmic or antiarrhythmic effects on OAP and triggered activity depending on the method of induction of OAP.
Regulation (effects) of OAP
2) Confidence 0.23 Published 1990 Journal J. Mol. Cell. Cardiol. Section Abstract Doc Link 1977923 Disease Relevance 0 Pain Relevance 0.18
We used OA as a control n-9 MUFA because this is the most abundant fatty acid in the diet [9].
Regulation (control) of OA
3) Confidence 0.06 Published 2007 Journal Lipids Section Body Doc Link PMC2039812 Disease Relevance 0.15 Pain Relevance 0.23

General Comments

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